149,99 €
A comprehensive summary of the state of the art in the management of fractures of the distal radius and carpal instability
Hand and wrist fractures account for millions of emergency room visits annually. The extraordinary importance of these structures in activities of daily living necessitates great surgical competence in repairing fractures, so as to preserve the vast range of motion and utility of this functional anatomic unit.
The management theory and techniques for these fractures have seen dramatic changes in the last few decades. This new volume brings together all currently established operative techniques for distal radius fractures, explained in detail and highly illustrated, step by step, with a wealth of brilliant figures and diagrams.
Key Features:
This book is ideal for all surgeons in training in orthopaedic surgery and in plastic surgery, and of benefit to experienced surgeons as well.
This book includes complimentary access to a digital copy on https://medone.thieme.com.
Das E-Book können Sie in Legimi-Apps oder einer beliebigen App lesen, die das folgende Format unterstützen:
Seitenzahl: 762
Veröffentlichungsjahr: 2019
Distal Radius Fractures and Carpal Instabilities
FESSH IFSSH 2019 Instructional Book
Editor-in-Chief:Francisco del Piñal, MD, PhDFormer Secretary General and Former President of EWASHand and Microvascular SurgeonMadrid and Santander, Spain
Co-Editors:Max Haerle, MD, PhDProfessorFormer General Secretary of FESSHFormer President of EWASDirector of Hand and Plastic Surgery DepartmentOrthopädische Klinik MarkgröningenMarkgröningen, Germany
Hermann Krimmer, MD, PhDProfessorChief of Hand CenterSt. Elisabeth HospitalRavensburg, Germany
645 illustrations
ThiemeStuttgart • New York • Delhi • Rio de Janeiro
Library of Congress Cataloging-in-Publication Data is available from the publisher.
© 2019. Thieme. All rights reserved.
Thieme Publishers StuttgartRüdigerstrasse 14, 70469 Stuttgart, Germany+49 [0]711 8931 421, [email protected]
Thieme Publishers New York333 Seventh Avenue, New York, NY 10001, USA+1-800-782-3488, [email protected]
Thieme Publishers DelhiA-12, Second Floor, Sector-2, Noida-201301Uttar Pradesh, India+91 120 45 566 00, [email protected]
Thieme Publishers Rio de Janeiro,Thieme Publicações Ltda.Edifício Rodolpho de Paoli, 25º andarAv. Nilo Peçanha, 50 – Sala 2508Rio de Janeiro 20020-906 Brasil+55 21 3172 2297 / +55 21 3172 1896
Cover design: Thieme Publishing GroupTypesetting by DiTech Process Solutions Pvt. Ltd., India
Printed in Germany by Aprinta GmbH, Wemding 5 4 3 2 1
ISBN 978-3-13-242379-4
Also available as an e-book:eISBN 978-3-13-242380-0
Important Note: Medicine is an ever-changing science undergoing continual development. Research and clinical experience are continually expanding our knowledge, in particular our knowledge of proper treatment and drug therapy. Insofar as this book mentions any dosage or application, readers may rest assured that the authors, editors, and publishers have made every effort to ensure that such references are in accordance with the state of knowledge at the time of production of the book.
Nevertheless, this does not involve, imply, or express any guarantee or responsibility on the part of the publishers with respect to any dosage instructions and forms of application stated in the book. Every user is requested to examine carefully the manufacturer’s leaflets accompanying each drug and to check, if necessary in consultation with a physician or specialist, whether the dosage schedules mentioned therein or the contraindications stated by the manufacturer differ from the statements made in the present book. Such examination is particularly important with drugs that are either rarely used or have been newly released on the market. Every dosage schedule or every form of application used is entirely at the user’s risk and responsibility. The authors and publishers request every user to report to the publishers any discrepancies or inaccuracies noticed.
Some of the product names, patents, and registered designs referred to in this book are in fact registered trademarks or proprietary names, even though specific reference to this fact is not always made in the text. Therefore, the appearance of a name without a designation as proprietary is not to be construed as a representation by the publisher that it is in the public domain.
This book, including all parts thereof, is legally protected by copyright. Any use, exploitation, or commercialization outside the narrow limits set by copyright legislation, without the publisher’s consent, is illegal and liable to prosecution. This applies in particular to photostat or mechanical reproduction, copying, or duplication of any kind, translating, preparation of microfilms, and electronic data processing and storage.
Preface
Contributors
1Anatomy of the Fracture
Simon MacLean, Gregory Bain
1.1 Introduction
1.2 Microanatomy of the Distal Radius: A Feat of Evolutionary Engineering
1.2.1 The Subchondral Bone Plate: the “Leaf Spring” of the Wrist
1.2.2 The Arch Bridge Concept
1.2.3 Multiple Rings Concept of the Wrist
1.3 The Role of the Carpal Ligaments
1.4 Fracture Initiation and Propagation
1.5 Scapholunate Dissociation and Two-Part Fractures of the Distal Radius
1.6 Volar Marginal Rim Fractures
1.7 A Biomechanical Model for Distal Radius Fracture
1.8 Summary
References
2Radiology of the Fractured Radius
Mark Ross, Patrick Groarke
2.1 Introduction
2.2 Plain Radiographs
2.2.1 Injury (Prereduction) Radiographs
2.2.2 Postreduction Radiographs
2.2.3 Parameters Can Change with Time
2.2.4 Radiograph of the Opposite Side
2.2.5 Posteroanterior View
2.2.6 Lateral View
2.3 Plain Radiographic Parameters
2.3.1 Posteroanterior View
2.3.2 Lateral View
2.4 Effect of Wrist Position on Carpal Indices
2.5 Reproducibility of Indices
2.6 Effect of Lateral Inclination on Volar Tilt
2.7 Intra-articular Components
2.8 Are the Parameters Reproducible?
2.9 Determination of Stability of Fracture Pattern and Adequacy of Reduction
2.9.1 Extra-articular Stability
2.9.2 Intra-articular Reduction
2.10 Other Signs on Radiographs
2.11 Frequency of Radiographic Assessment
2.12 Classification of Distal Radius Fractures
2.13 Computed Tomography
2.13.1 Sagittal Computed Tomography
2.13.2 Coronal Computed Tomography
2.13.3 Axial Computed Tomography
2.13.4 Computed Tomography and DRUJ Stability
2.13.5 Three-Dimensional Reconstructions
2.14 Magnetic Resonance Imaging and Distal Radius Fractures
2.15 Summary
2.15.1 Plain Radiographs
2.15.2 Computed Tomography Scans
2.15.3 Magnetic Resonance Imaging Scans
References
3Patient–Accident–Fracture Classification of Acute Distal Radius Fractures in Adults
Guillaume Herzberg, Thais Galissard, Marion Burnier
3.1 Introduction
3.2 Material and Methods
3.2.1 Patient
3.2.2 Accident
3.2.3 Fracture
3.3 Results and Discussion
3.3.1 Patients 1–1 (Dependent, Minimal Functional Needs) with AO Type “A” or “C” Fractures
3.3.2 Patients 2–2 (Independent Patients with Comorbidities, Intermediate Functional Needs) with AO Type “A” Extra-articular Fractures
3.3.3 Patients 2–2 (Independent Patients with Comorbidities, Intermediate Functional Needs) with AO Type “C” Intra-articular Fractures
3.3.4 Patients 3–3 (Patient with Normal General Health Status and Maximal Functional Needs) with AO Type “A” Extra-articular Fractures
3.3.5 Patients 3–3 (Patient with Normal General Health Status and Maximal Functional Needs) with AO Type “C” Intra-articular Fractures
3.3.6 Patients 3–3 (Patient with Normal General Health Status and Maximal Functional Needs) with AO Type “B” Partial Articular Fractures
3.4 Conclusion
References
4Distal Radius Fracture: The Evidence
Tracy Webber, Tamara D. Rozental
4.1 Introduction
4.2 Methods
4.3 Nonoperative Fracture Treatment
4.4 Surgery versus Immobilization for Displaced Distal Radius Fracture
4.5 The Management of Elderly Patients
4.6 Operative Treatment for Displaced Distal Radius Fracture: Method of Fixation
4.6.1 Bridging versus Nonbridging External Fixation
4.6.2 Closed Reduction Percutaneous Pinning versus Open Reduction Internal Fixation
4.6.3 Closed Reduction Percutaneous Pinning versus External Fixation
4.6.4 Open Reduction Internal Fixation versus External Fixation
4.6.5 Implant Type
4.6.6 Management of the Pronator Quadratus
4.7 Postoperative Rehabilitation
4.8 Management of Complications
4.9 Treatment for Osteoporosis
4.10 Conclusion
References
5Orthopaedic Treatment: When?
David Warwick, Oliver Townsend
5.1When the Evidence and Benefits of Nonoperative Treatment Have Been Considered
5.1.1 Why Surgery Is Not Usually Needed
5.1.2 Nonoperative Treatment Is Usually Effective
5.1.3 Surgery Carries Risk
5.1.4 Risks of Nonoperative Intervention
5.2When the Literature Recommendations Have Been Considered
5.2.1 Drawbacks of the Literature
5.2.2 Authors’ Recommendations
5.2.3 Consensus Review
5.3When Age Has Been Considered
5.3.1 Older Age
5.3.2 Younger Age
5.4When There Is a Predictable Risk of Poor Outcome
5.4.1 Exclusion Bias
5.4.2 Distal Radioulnar Joint Incongruity
5.4.3 Adaptive Midcarpal Malalignment
5.4.4 Positive Ulnar Variance
5.4.5 Fracture Dislocation
5.5When There Is a Predictable Risk of Instability
5.5.1 Instability
5.5.2 Prediction of Instability
5.6When There Is a Predictable Risk of Symptomatic Arthritis
5.6.1 Does Arthritis Really Matter in the Hand and Wrist?
5.6.2 How Often Does a Hand Surgeon Actually See Arthritis after a Distal Radius Fracture?
5.6.3 Literature Evidence
5.7When the Appropriate Method Is Available
5.8When the Patient Understands the Options and the Risks
5.8.1 The Patient’s Rights
5.8.2 Honest Consent
5.8.3 Personalized Treatment
5.9When the Patient Prefers to Have Urgent Surgery that May Not Be Needed rather than Wait for a Corrective Osteotomy
5.9.1 Natural History of Recovery after Distal Radius Fracture
5.9.2 Managing Uncertainty
5.9.3 Rescuing a Wrong Decision with Corrective Osteotomy
5.10When the Patient Wants Early Return of Function
5.11When Cost-Benefit Analysis Supports Surgery
5.12 Conclusion
References
6Is There a Role for External Fixation with or without Kirschner Wires?
Frédéric Schuind
6.1 Introduction
6.2 Indications and Contraindications
6.3 Surgical Techniques
6.4 Prevention of Redisplacement
6.5 Prevention of Other Complications of External Fixation
6.6 Results
6.7 Conclusion
References
7Volar Locking Plates: Basic Concepts
Hermann Krimmer
7.1 Introduction
7.2 Locking Mechanisms
7.3 Indications
7.3.1 Plate Design and Plate Position
7.3.2 Fracture-Specific Plate Selection
7.4 Surgical Technique
7.5 Pitfalls
7.5.1 Tendon Ruptures
7.5.2 Secondary Subsidence
References
8Intramedullary Devices
Stephanie Malliaris, Scott Wolfe
8.1 Introduction
8.2 Indications
8.3 Intramedullary Nail: Surgical Technique
8.4 Intramedullary Nail: Outcomes
8.5 T-Pin: Surgical Technique
8.6 T-Pin: Outcomes
8.7 Distal Radius System Intramedullary Cage: Surgical Technique
8.8 Distal Radius System Intramedullary Cage: Outcomes
8.9 Pitfalls and Contraindications
8.10 Conclusion
References
9Mini Approaches
Gustavo Mantovani Ruggiero
9.1 Introduction
9.2 Volar Minimally Invasive Plate Osteosynthesis History
9.3 Indications
9.4 Surgical Technique
9.4.1 Single Longitudinal Incision
9.4.2 Transverse Incision
9.5 Evolution of Distal Radius Fractures Minimally Invasive Plate Osteosynthesis
9.5.1 Special Plates
9.6 Results, Clinical Examples, and Unusual Minimally Invasive Plate Osteosynthesis Cases
9.7 Pitfalls and Contraindications
9.8 Conclusions
References
10Spanning Plates
Mitchell G. Eichhorn, Scott G. Edwards
10.1 Introduction
10.2 Indications
10.3 Surgical Technique
10.3.1 Third Metacarpal Technique
10.3.2 Second Metacarpal Technique
10.4 Results
10.5 Pitfalls and Contraindications
10.6 Conclusion
References
11Arthroscopic Management
Yukio Abe
11.1 Introduction
11.2 Indications
11.3 Surgical Technique
11.3.1 Preoperative Planning
11.3.2 Preparation and Patient Positioning
11.3.3 Surgical Approach
11.3.4 Postoperative Care
11.4 Results
11.4.1 The Advantages of Wrist Arthroscopy
11.4.2 Outcomes
11.5 Complications
11.6 Conclusion
References
12Anterior Rim Fractures
Jorge L. Orbay, Gabriel Pertierra
12.1 Introduction
12.1.1 Background
12.1.2 Classification
12.1.3 Anatomy and Biomechanics
12.2 Indications
12.3 Surgical Technique
12.3.1 Salvage of a Collapsed VMF
12.4 Results
12.5 Pitfalls and Contraindications
12.6 Conclusion
References
13Dorsal Rim Distal Radius Fractures with Radiocarpal Fracture-Dislocation
Rohit Garg, Jesse Jupiter
13.1 Introduction
13.2 Indications
13.3 Surgical Technique
13.3.1 Case 1
13.3.2 Case 2
13.3.3 Case 3
13.3.4 Case 4
13.4 Results
13.5 Conclusion
References
14Multiplanar Fixation in Severe Articular Fractures
Peter C. Rhee, Alexander Y. Shin
14.1 Introduction
14.1.1 Understanding the Fracture Characteristics
14.1.2 Limitations of Volar Locking Plate Fixation
14.1.3 Rationale for Fragment-Specific Fixation
14.2 Indications
14.3 Surgical Technique (Authors’ Preferred)
14.3.1 Preoperative Planning
14.3.2 Sequence of Reconstruction
14.3.3 Management of the Volar Rim Fragment
14.3.4 Management of the Dorsal Ulnar Corner Fragment
14.3.5 Management of Free Intra-articular Fragments
14.3.6 Management of the Dorsal Wall Fragment
14.3.7 Management of the Radial Column Fragment
14.3.8 Finishing Steps
14.4 Results for Multiplanar Plate Fixation with Fragment-Specific Implants
14.4.1 Functional Outcomes
14.4.2 Radiographic Outcomes
14.4.3 Complications
14.4.4 Comparison of Fragment-Specific to Volar Locking Plate Fixation
14.5 Pitfalls and Contraindications
14.6 Conclusion
References
15Management of Wrist Open Fractures and Bone Defects
Rames Mattar Junior, Emygdio Jose Leomil de Paula, Luciano Ruiz Torres, Tiago Guedes da Motta Mattar, Gustavo Bersani Silva
15.1 Introduction
15.2 Indications
15.3 Surgical Technique
15.3.1 Fracture Management
15.3.2 Soft Tissue Repair
15.3.3 Nerve Injuries
15.3.4 Tendons
15.3.5 Amputations and Devascularizations
15.4 Results
15.5 Pitfalls
15.6 Conclusion
References
16Radiocarpal Dislocation
Mark Henry
16.1 Introduction
16.2 Indications
16.3 Surgical Technique
16.4 Results
16.5 Pitfalls and Contraindications
References
17Common Errors of Volar Plate Fixation
Robert J. Medoff, James M. Saucedo
17.1 Background
17.2 Errors of Surgical Exposure
17.3 Hardware-Related Errors
17.4 Tendon Complications
17.4.1 Flexor Tendon Rupture and Irritation
17.4.2 Extensor Tendon Injury and Rupture
17.5 Inadequate Reduction and Fixation
17.6 Conclusion
References
18Distal Ulna Fractures
Christopher Klifto, David Ruch
18.1 Introduction
18.2 Indications
18.2.1 Ulnar Styloid Fractures/Nonunions
18.3 Surgical Technique
18.3.1 Approach
18.3.2 Ulnar Styloid Fractures Technique
18.3.3 Ulnar Head Fractures
18.3.4 Comminuted Intra-articular Distal Ulnar Fractures
18.4 Results
18.5 Pitfalls
18.6 Conclusion
References
19Distal Radioulnar Joint Instability Associated with Distal Radius Fractures
Shohei Omokawa, Takamasa Shimizu, Kenji Kawamura, Tadanobu Onishi
19.1 Introduction
19.2 Pathomechanics of DRUJ Instability
19.2.1 Metaphyseal Fracture Displacement
19.2.2 Disruptions Accompanying TFCC (Radioulnar Ligament) Tears
19.2.3 Ulnar Styloid Fractures
19.2.4 Intra-articular Displacement of the Sigmoid Notch
19.3 Case Presentation
19.4 Diagnosis of Accompanying TFCC Tears and DRUJ Instability
19.4.1 Preoperative Assessment
19.4.2 Intraoperative Assessment
19.4.3 DRUJ Arthrography
19.5 Treatment for TFCC Tears and DRUJ Instability
19.5.1 Authors’ Preferred Method
19.6 Summary
References
20Distal Radius Fracture in the Elderly
Rohit Arora, Markus Gabl
20.1 Introduction
20.2 Indications
20.3 Treatment Options
20.3.1 Closed Reduction and Cast Immobilization
20.3.2 Closed Reduction and Percutaneous Pinning
20.3.3 External Fixation
20.3.4 Volar Locking Plate Fixation
20.3.5 Distal Radius Arthroplasty
20.3.6 Initial Shortening and Palmar Plate Fixation with Primary Sauve-Kapandji Procedure
20.4 Complications
20.5 Conclusion
20.6 Our Algorithm of Treatment
20.7 Initial Treatment
References
21Extra-articular Malunion
Karl-Josef Prommersberger
21.1 Introduction
21.1.1 Biomechanics of Distal Radial Malunion
21.1.2 Treatment Options
21.2 Indications and Contraindications
21.3 Surgical Technique
21.3.1 Timing for Radial Correction Osteotomy in an Extra-articular Malunion
21.3.2 Preoperative Work-up
21.3.3 Technique
21.4 Results
21.4.1 Own Results
21.5 Pitfalls
21.6 Conclusion
References
22Arthroscopic-Guided Osteotomy for Intra-articular Malunion
Francisco del Piñal
22.1 Introduction
22.2 Indications and Contraindications
22.3 Surgical Technique
References
23Newer Technologies on Managing Distal Radius Fractures
Ladislav Nagy
23.1 Introduction
23.2 Indications
23.3 Surgical Technique
23.3.1 Extra-articular Malunion
23.3.2 Intra-articular Malunion
23.3.3 Intra-articular Fractures of the Distal Radius
23.4 Results
23.5 Pitfalls and Contraindications
23.6 Conclusion
References
24Distal Radius in Children and Growth Disturbances
Alexandria L. Case, Joshua M. Abzug
24.1 Distal Radius Fractures in Children: Introduction
24.1.1 Extraphyseal Fractures
24.1.2 Physeal Fractures
24.1.3 Indications and Contraindications
24.1.4 Surgical Technique
24.1.5 Results
24.1.6 Pitfalls
24.2 Growth Disturbances Following Distal Radius Fractures: Introduction
24.2.1 Indications and Contraindications
24.2.2 Surgical Techniques
24.2.3 Results
24.2.4 Pitfalls
24.3 Conclusion
References
25Open Surgery for Chronic Scapholunate Injury
Dirck Ananos, Marc Garcia-Elias
25.1 Introduction
25.1.1 Terminology
25.1.2 Ligaments Involved in Scapholunate Instability
25.2 Indications
25.3 Surgical Technique
25.4 Results, Pearls, and Tips
25.4.1 Case Example
25.4.2 Pitfalls and Contraindications
25.5 Conclusion
References
26Arthroscopic Scapholunate Repair
Max Haerle, Christophe Mathoulin
26.1 Introduction
26.2 Indications
26.3 Surgical Technique
26.4 Results
26.5 Pitfalls and Contraindications
26.6 Conclusion
References
27Treatment of Lunotriquetral Injuries
Benjamin F. Plucknette, Haroon M. Hussain, Lee Osterman
27.1 Anatomy
27.2 Diagnosis and Classification
27.3 Treatment of Acute LT Injuries
27.3.1 Nonoperative Management
27.3.2 Operative Management
27.4 Treatment of Chronic Lunotriquetral Injuries
References
28Arthroscopic Management of Perilunate Dislocation and Fracture Dislocation
Jae Woo Shim, Jong-Pil Kim, Min Jong Park
28.1 Introduction
28.2 Indications
28.3 Surgical Technique
28.4 Results
28.4.1 Clinical Results
28.4.2 Radiological Results
28.5 Complications
28.6 Pitfalls and Contraindications
28.7 Conclusion
References
29Radiocarpal Pain and Stiffness
Christoph Pezzei, Stefan Quadlbauer
29.1 Introduction
29.2 Surgical Techniques
29.2.1 Denervation of the Wrist
29.2.2 Wrist Arthrolysis
29.2.3 Partial Wrist Arthrodesis
29.2.4 Wrist Arthroplasty
29.2.5 Total Wrist Arthrodesis
29.3 Conclusion
References
30Ulnar Pain and Pronosupination Losses
Riccardo Luchetti, Andrea Atzei
30.1 Introduction
30.2 Indication
30.3 Diagnostic Imaging
30.3.1 Radiography
30.3.2 CT Scan
30.3.3 Magnetic Resonance Imaging
30.3.4 Arthrography, ArthroCT, and ArthroMRI
30.4 Surgical Options
30.4.1 Open Arthrolysis
30.4.2 Arthroscopic Arthrolysis
30.5 Results
30.6 Contraindications
30.7 Conclusion
30.8 Types of Painful DRUJ Rigidity
30.8.1 Dislocation or Dorsal Subluxation and Positive Ulna Variation Secondary to Distal Radius Malunion
30.8.2 Loss of Pronosupination due to Screws Interposed into the DRUJ
30.8.3 Interposition of the Extensor Digiti Minimi
30.8.4 Ulna Malunion
References
31Chronic Distal Radioulnar Joint Instability
Michael C. K. Mak, Pak-Cheong Ho
31.1 Introduction
31.1.1 Distal Radioulnar Joint Anatomy and Its Stabilizers
31.2 Indications
31.2.1 Correction of Skeletal Deformity
31.2.2 Triangular Fibrocartilage Complex Reconstruction
31.3 Surgical Technique
31.3.1 Setup and Instruments
31.4 Rehabilitation
31.5 Outcomes and Complications
31.6 Conclusion
References
Index
It is a great privilege that the International Federation of Societies for Surgery of the Hand (IFSSH) and the Federation of European Societies for Surgery of the Hand (FESSH) have considered me as editor-in-chief of this book on the burning issue of distal radius fractures and ligamentous injuries.
Management of distal radius fractures and wrist ligamentous injuries has seen dramatic changes in the last few decades. For those of us who have lived this on the front line, it has been a very exciting experience. Beginning from casts, external fixateurs, intramedullary devices, volar or dorsal plates, and finally to arthroscopy, all have contributed to making it possible to provide our patients with outstanding results. One thing we have learned is that no one method can be used for all injuries, as the personality of each fracture or ligamentous injury demand a different approach. For this reason, every available treatment has its own space in our armamentarium.
Fully conscious of this, we have compiled here an up-to-date selection of the techniques available to tackle all injury types. The topics have been carefully selected to cover the subjects of greatest interest or higher impact. In this endeavor, we have had the fortune to count on world-renowned surgeons, who have championed the changes we are now using in our everyday practice. Some chapters provide new answers or refinements to recurrent problems.
This book will hopefully improve the care of our patients and, furthermore, inspire the creativity of future generations of surgeons.
Finally, I would like to thank my co-editors and all the authors who have devoted their precious time to sharing their knowledge with us.
Paco PiñalMadrid, 2019
Yukio Abe, MD, PhD
Director
Department of Orthopaedic Surgery
Saiseikai Shimonoseki General Hospital
Shimonoseki, Japan
Joshua M. Abzug, MD
Associate Professor
Departments of Orthopedics and Pediatrics
University of Maryland School of Medicine
Director of Pediatric Orthopedics
University of Maryland Medical Center
Deputy Surgeon-in-Chief
University of Maryland Children‘s Hospital
Baltimore, Maryland, USA
Dirck Ananos, MD
Fellow
Kaplan Hand Institute
Barcelona, Spain
Rohit Arora, MD
Department of Trauma Surgery
Medical University Innsbruck
Innsbruck, Austria
Andrea Atzei, MD
Fenice Hand Surgery and Rehabilitation Team
Treviso, Italy
Gregory Bain, MD
APWA President
Professor of Upper Limb and Research
Department of Orthopaedic Surgery
Flinders University
Adelaide, South Australia, Australia
Marion Burnier, MD
Wrist Surgery Unit
Department of Orthopaedics
Claude-Bernard Lyon 1 University
Herriot Hospital
Lyon, France
Alexandria L. Case, MD
Clinical Research Coordinator
Department of Orthopedics
University of Maryland School of Medicine
Baltimore, Maryland, USA
Scott G. Edwards, MD
Chief of Hand and Upper Extremity Surgery
Banner University Medical Center
The CORE Institute Specialty Hospital
Phoenix, Arizona, USA
Mitchell G. Eichhorn, MD
Hand Surgery Fellow
University of Arizona Banner Hand Surgery Fellowship
Phoenix, Arizona, USA
Markus Gabl, MD
Department of Trauma Surgery
Medical University Innsbruck
Innsbruck, Austria
Thais Galissard, MD
Wrist Surgery Unit
Department of Orthopaedics
Claude-Bernard Lyon 1 University
Herriot Hospital
Lyon, France
Marc Garcia-Elias, MD, PhD
Consultant and Co-Founder
Kaplan Hand Institute
Barcelona, Spain
Honorary Consultant
Pulvertaft Hand Center
Derby, UK
Rohit Garg, MBBS
Hand and Upper Extremity Orthopaedic Surgeon
Massachusetts General Hospital
Boston, Massachusetts, USA
Patrick Groarke, MD
Brisbane Hand and Upper Limb Research Institute
Brisbane Private Hospital
Brisbane, Queensland, Australia
Orthopaedic Department
Princess Alexandra Hospital
Woolloongabba, Queensland, Australia
Max Haerle, MD, PhD
Professor
Former General Secretary of FESSH
Former President of EWAS
Director of Hand and Plastic Surgery Department
Orthopädische Klinik Markgröningen
Markgröningen, Germany
Mark Henry, MD
Practicing Hand Surgeon
Hand and Wrist Center of Houston
Houston, Texas, USA
Guillaume Herzberg, MD, PhD
Professor of Orthopaedic Surgery
Lyon Claude Bernard University
Herriot Hospital
Lyon, France
Pak-Cheong Ho, MD, MBBS, FRCS, FHKCOS, FHKAM(Ortho)
EWAS Former President
APWA Founder and Former President
Department of Orthopaedics and Traumatology
Prince of Wales Hospital
Chinese University of Hong Kong
Hong Kong, SAR China
Haroon M. Hussain, MD
Greater Washington Orthopaedic Group PA
Rockville, Maryland, USA
Rames Mattar Junior, MD
Associate Professor
Department of Orthopedic
Hand and Microsurgery Unit
University Of São Paulo
São Paulo, Brazil
Jesse Jupiter, MD
Hansjorg Wyss AO Professor
Department of Orthopedic Surgery
Massachusetts General Hospital
Harvard Medical School
Boston, Massachusetts, USA
Kenji Kawamura, MD, PhD
Tamai Susumu Memorial Hand and Extremity Trauma Center
Nara Medical University
Kashihara, Japan
Jong-Pil Kim, MD, PhD
Professor
Department of Orthopedic Surgery
Dankook University College of Medicine
Cheonan, South Korea
Christopher Klifto, MD
Assistant Professor
Department of Orthopaedic Surgery
Hand Division
Duke University Medical Center
Durham, North Carolina, USA
Hermann Krimmer, MD, PhD
Professor
Chief of Hand Center
St. Elisabeth Hospital
Ravensburg, Germany
Riccardo Luchetti, MD
Rimini Hand Surgery and Rehabilitation Center
Rimini, Italy
Simon MacLean, MBChB, FRCS(Tr&Orth), PGDipCE
Consultant Orthopaedic and Upper Limb Surgeon
Tauranga Hospital, BOPDHB
Tauranga, New Zealand
Michael C. K. Mak, MD
Division of Hand and Microsurgery
Department of Orthopaedics and Traumatology
Prince of Wales Hospital
Chinese University of Hong Kong
Hong Kong, SAR China
Stephanie Malliaris, MD
Assistant Professor
Plastic and Reconstructive Surgery
University of Colorado School of Medicine
Attending Surgeon
Denver Health Medical Center
Denver, Colorado, USA
Christophe Mathoulin, MD, FMH
Vice-President
Institut de la Main
Founder and Honorary Chairman
European (International) Wrist Arthroscopy Society (EWAS - IWAS)
Founder and Chairman
International Wrist Center–Wrist Clinic
Clinique Bizet
Paris, France
Tiago Guedes da Motta Mattar, MD
Department of Orthopedic
Hand and Microsurgery Unit
University of São Paulo
São Paulo, Brazil
Robert J. Medoff, MD
Assistant Professor
Department of Surgery
University of Hawaii
Honolulu, Hawaii, USA
Ladislav Nagy, MD
Professor
Hand Surgery Division
University Clinic Balgrist
Zürich, Switzerland
Shohei Omokawa, MD, PhD
Department of Hand Surgery
Nara Medical University
Kashihara, Japan
Tadanobu Onishi, MD
Department of Orthopedic Surgery
Nara Medical University
Kashihara, Japan
Jorge L. Orbay, MD
Hand & Upper Extremity Surgeon
The Miami Hand & Upper Extremity Institute
Miami, Florida, USA
Lee Osterman, MD
Professor, Hand and Orthopedic Surgery
Thomas Jefferson University
President
Philadelphia Hand to Shoulder Center
Philadelphia, Pennsylvania, USA
Min Jong Park, MD
Department of Orthopaedic Surgery
Samsung Medical Center
Sungkyunkwan University School of Medicine
Seoul, South Korea
Emygdio Jose Leomil de Paula, MD, PhD
Department of Orthopedic
Hand and Microsurgery Unit
University Of São Paulo
São Paulo, Brazil
Gabriel Pertierra, BA
The Miami Hand & Upper Extremity Institute
Miami, Florida, USA
Christoph Pezzei, MD
Department of Traumatology
AUVA Trauma Hospital Lorenz Böhler–European Hand Trauma Center
Vienna, Austria
Francisco del Piñal, MD, PhD
Former Secretary General and Former President of EWAS
Hand and Microvascular Surgeon
Madrid and Santander, Spain
Benjamin F. Plucknette, DO, DPT
Orthopaedic, Hand, and Microvascular Surgeon
Department of Orthopaedic Surgery
San Antonio Military Medical Center
JBSA-Fort Sam Houston, Texas, USA
Karl-Josef Prommersberger, MD
Professor
Clinic for Hand Surgery
Rhön Klinikum AG
Salzburger Leite
Bad Neustadt an der Saale, Germany
Stefan Quadlbauer, MD
AUVA Trauma Hospital Lorenz Böhler–European Hand Trauma Center
Ludwig Boltzmann Institute for Experimental and Clinical Traumatology
AUVA Research Center
Austrian Cluster for Tissue Regeneration
Vienna, Austria
Peter C. Rhee, DO, MS
Orthopedic Surgery
Mayo Clinic
Rochester, Minnesota, USA
Mark Ross, MD
Brisbane Hand and Upper Limb Research Institute
Brisbane Private Hospital
Brisbane, Queensland, Australia
Orthopaedic Department
Princess Alexandra Hospital
Woolloongabba, Queensland, Australia
School of Medicine
The University of Queensland
St Lucia, Queensland, Australia
Tamara D. Rozental, MD
Chief, Hand and Upper Extremity Surgery
Associate Professor
Department of Orthopaedic Surgery
Beth Israel Deaconess Medical Center
Harvard Medical School
Boston, Massachusetts, USA
David Ruch, MD
Professor and Chief of Division of Hand and Microvascular Surgery
Adjunct Professor of Plastic and Reconstructive Surgery
Duke University Medical Center
Durham, North Carolina, USA
Gustavo Mantovani Ruggiero, MD
São Paulo Hand Center
Hospital Beneficência Portuguesa de São Paulo
São Paulo, Brazil
Hand Surgery Department
Plastic Surgery School
Ospedale San Giuseppe
Università Degli Studi Di Milano
Milan, Italy
James M. Saucedo, MD
The Hand Center of San Antonio
Adjunct Assistant Professor
Department of Orthopaedics
University of Texas Health San Antonio
San Antonio, Texas, USA
Frédéric Schuind, MD, PhD
Full Professor
Université libre de Bruxelles
Head, Department of Orthopaedics and Traumatology
Erasme University Hospital
Brussels, Belgium
Jae Woo Shim, MD
Department of Orthopaedic Surgery
Samsung Medical Center
Sungkyunkwan University School of Medicine
Seoul, South Korea
Takamasa Shimizu, MD, PhD
Department of Orthopedic Surgery
Nara Medical University
Kashihara, Japan
Alexander Y. Shin, MD
Orthopedic Surgery
Mayo Clinic
Rochester, Minnesota, USA
Gustavo Bersani Silva, MD
Department of Orthopedic
Hand and Microsurgery Unit
University Of São Paulo
São Paulo, Brazil
Luciano Ruiz Torres, MD
Department of Orthopedic
Hand and Microsurgery Unit
University of São Paulo
São Paulo, Brazil
Oliver Townsend, BSc, MBBS, MRCS
Core Surgical Fellow
Southampton General Hospital
University Hospital Southampton
Southampton, UK
David Warwick, MD, FRCSOrth, Eur Dip Hand Surg
Professor and Consultant Hand Surgeon
University Hospital Southampton
Southampton, UK
Tracy Webber, MD, BIDMC
Harvard Orthopaedic Hand Fellowship
Department of Orthopaedic Surgery
Beth Israel Deaconess Medical Center
Harvard Medical School
Boston, Massachusetts, USA
Scott Wolfe, MD
Professor
Department of Orthopaedic Surgery
Hospital for Special Surgery
Weill Medical College of Cornell University
New York, New York, USA
Simon MacLean, Gregory Bain
Abstract
This chapter discusses the importance of the anatomy of distal radius in relation to fracture. First, we describe the microanatomy of the distal radius. Its microanatomy resembles that seen in everyday engineering structures; the subchondral bone plate and arrangement of trabeculae enabling the wrist to handle high multidirectional loads. Stability of the wrist is achieved by multiple ligamentous rings that confer stability within and between the rows and columns of the wrist. Ligamentous insertions play an important role in fracture morphology, both initiation and propagation of the fracture lines.
Hand position at impact determines the position of the carpus on the distal radius. When a line of differential load occurs in the scapholunate interval, scapholunate ligament injury (SLI) may result. SLI is associated with specific fracture types. “Unmasking” with scapholunate diastasis occurs when there is compromise to the secondary stabilizers.
The volar marginal rim fracture represents an important subset of fractures. These are associated with a higher rate of carpal ligamentous injury and fixation failure. Distal radius plate design has evolved to attempt to capture this fragment.
Lastly, we present a biomechanical model for distal radius fracture.
Keywords: distal radius fracture, microstructure, scapholunate injury, volar rim fracture, radiocarpal ligaments
Distal radius fractures (DRFs) are one of the most commonly treated injuries in orthopaedic practice. A bimodal distribution exists; in younger patients, caused by high-energy and in older patients, caused by low-energy falls. Osteoporotic DRFs reflect a change in the microarchitecture of the distal radius with aging and are a predictor for subsequent fracture of long bones. Classification systems have attempted to provide clarity to the morphology and treatment of these injuries.
With an improved understanding of the anatomy of distal radius, we are better equipped to treat the patient, respecting not just the fracture components but also the contribution of the surrounding ligaments, carpal bones, and other associated injuries. This chapter will explore the importance of each component and its interaction in the mechanism of fracture. First, we will study the microanatomy of the distal radius; and then we will focus on fracture initiation and the role of the carpus; and third, the role of the carpal ligaments and fracture propagation. We will look at the specific anatomy of the volar rim fracture. We will propose a biomechanical model for DRF.
Singh reported the trabecular structure of the proximal femur and changed our approach to the understanding and management of these injuries.1 The microstructure of the distal radius assists in understanding its behavior under load. We performed an analysis of the architecture of cadaveric distal radii on micro-computed tomography (CT)2,3 and found it resembled many of the engineering concepts in everyday man-made structures. Interestingly, an engineer once advised us that he knew when he had the design correct and when it resembled the structures identified in nature.
The subchondral bone plate is a 2-mm-thick multilaminar plate that absorbs impact and transmits the load to the radial metaphysis. The superficial “primary” bone plate bridges the entire articular margin. Multiple deeper layers resemble a leaf spring used in the suspension of heavy motor vehicles.
Between these laminae, and connecting them, lie intralaminar struts. These are initially perpendicular to the joint line, but more proximally align with the radial shaft. Voids between the struts and laminae absorb impact and enable vessels to perforate. The struts between the laminae are mini “I-beams,” which make the structure strong but still enable it to bend. Engineers refer to this multilayered lamina construct as a “honeycomb sandwich panel.”
With physiological wrist extension, the volar capsule becomes taut, and the scaphoid and lunate load the distal radius subchondral bone plate.
The multilayered subchondral bone plate resists buckling and transmits the load to the intermediate trabeculae and then the metaphyseal arches (▶Fig. 1.1).
On a magnified view, areas can be seen where these trabeculae coalesce to form a rod and extend as longitudinal ridges down the diaphysis (▶Fig. 1.2). These align in the direction of load through the cortical bone and reinforce the radius, similar to the longerons in a plane’s fuselage (▶Fig. 1.3), and prevent torsional failure and buckling.
The load bearing area of the lunate is very volar. The sagittal images demonstrate the thick volar trabecular columns spanning to the volar cortex of the metaphyseal radius (▶Fig. 1.4). This explains the devastating effect of Barton volar lip fracture, as the carpus will simply dislocate volarly.
Fig. 1.1 Anatomy of the subchondral bone plate of the distal radius.(Copyright © Gregory Bain, MD)
Fig. 1.2 The radial styloid cortex is quite thin but is reinforced by bracing trabeculae. The trabeculae are thin sheets of bone, which are designed to transmit load. There the sheets meet and coalesce into rods, which then become the ridges on the endosteal cortex.(Copyright © Gregory Bain, MD)
Further dorsal on the lunate facet, trabeculae course dorsally forming a curve in the shape of a gothic arch, with an underlying intramedullary “vault.” From medial to lateral through the distal radius, these gothic arches are in series—connected by interarch struts. In normal bone under normal loads, the parabolic shape of the arch transfers loads in a longitudinal and lateral direction, to the base of the arch without creating tension (▶Fig. 1.4).
At the base of each arch, the trabeculae merge with the cortex, which then becomes thickened—therefore buttressing the arch. In contrast, at the articular margin, the cortex is thin. It acts to suspend the subchondral bone plate and serves as a site for ligament attachment, rather than to bear the load.
The microstructure of the distal radius resembles an arch bridge with the following equivalent: deck—SBP, intermediate struts—intermediate trabeculae, arches—arches, and bridge foundation—cortex. The deck is a tightly held lattice with multiple “I-beams” in a multilayer sandwich panel construct. This resists buckling, absorbs impact, and takes the entire load (▶Fig. 1.5).
The arches and intermediate struts are a semiflexible construct, which distributes compressive forces from the deck to the base (diaphysis).4 The orientation of the microstructure ensures that forces are distributed in compression rather than tension (where the bone is weakest).
Force transfer from the hand to the radius at the time of DRF involves the transmission of force through the three columns of the wrist. The three columns are bound by a series of ligamentous rings. These ligamentous rings provide stability within and between the proximal and distal rows of the wrist (▶Fig. 1.6).
The distal radioulnar joint is bound by a fibroligamentous ring. Disruption of this at the time of DRF can lead to dislocation of this joint and distal radioulnar instability, if not addressed at the time of surgery.
Within the proximal carpal row, interosseous ligaments on the volar and dorsal aspects of the scapholunate and lunotriquetral joints form a ring. Transmission of force from the central column to the scaphoid, lunate, or both can lead to a disruption of these ligaments and a “greater arc” injury or an impaction fracture of the distal radius.
Fig. 1.3 Longitudinal ridges in the diaphysis of the distal radius resembling a plane’s fuselage.(Copyright © Gregory Bain, MD)
Fig. 1.4 The sagittal microanatomy of the distal radius resembling a gothic arch.(Copyright © Gregory Bain, MD)
The distal carpal row is tightly bound by interosseous ligaments, allowing for minimal motion within the row. Disruption of this ring rarely occurs at the time of DRF but is seen with high-energy axial fracture-dislocations of the carpus.
A series of ligaments connect the rows of the wrist. Kuhlmann ring refers to the volar radiotriquetral ligament and the dorsal radiocarpal ligament (DRC) complexes.5 Disruption of this ring in complex high-energy DRFs can lead to ulnar translocation of the carpus. The DRC also acts as a secondary stabilizer to the scapholunate joint.
The scaphotrapeziotrapezoid (STT) ligament stabilizes the scaphoid to the distal carpal row. As a secondary stabilizer of the scapholunate joint, disruption of this complex can lead to scapholunate diastasis and dorsal intercalated segmental instability (DISI) deformity.
The dorsal and volar ligamentous complexes surrounding the wrist are anatomically and functionally distinct. The stout volar capsular ligaments are a complex series of condensations in the thick volar capsule.6,7 In contrast, there are only two named dorsal ligaments: the dorsal radiocarpal and dorsal intercarpal (DIC) ligaments. The remainder of the dorsal capsule is extremely elastic.
Fig. 1.5 The microstructure of the distal radius resembling an arch bridge.(Copyright © Gregory Bain, MD)
Fig. 1.6 The “multiple rings” of the wrist.(Copyright © Gregory Bain, MD)
Both Melone and Medoff described the importance of the ligamentous attachments of the distal radius.8–11 Melone highlighted the role of the two medial fragments for the articular function: “the medial complex” and its strong ligamentous attachments.10 Medoff recognized the contributions of ligaments to fracture displacement, and described radiocarpal instability and the contribution of ligament avulsion to the creation of “rim” fragments, leading to catastrophic failure of fixation.11
In our study by Mandziak et al,12 we performed CT mapping of 100 distal radius intra-articular fractures and identified that fracture lines were significantly more likely to occur between the ligament insertions (▶Fig. 1.7).6,7 In low-energy injuries, the fractures almost never include the ligament attachments but are between the ligaments. High-energy injuries are more random and reflect the forces placed on the wrist.
The ligaments are designed to resist tension, which is maximal at the extremes of motion. Bone is designed to take the compressive load and fail in tension. The ligaments play two key roles in the mechanism of fracture.
Our study, however, suggests that ligament insertion points may be protective for DRFs, as most fracture lines avoided these sites. This would correlate with the role of ligamentotaxis with fracture reduction. In the exception of the isolated “die-punch” fracture, ligamentotaxis reduces distal radius morphology. Even in cases of extreme comminution, an initial almost anatomical reduction may be possible by the creation of radiocarpal ligament tension across the joint. This would not be possible in the setting of radiocarpal ligament avulsion. Failure of the microarchitecture and the intraosseous arches may lead to later collapse—hence the high rate of redisplacement in fractures with comminution in osteoporotic bone.13 “Bridge plating” is an example of ligamentotaxis in the case of articular comminution. The “bridge” is used to maintain ligamentotaxis and neutralize deforming forces before osseous union occurs.
Fig. 1.7 Fractures most commonly involve the interligamentous zones 10, 2, and 6.(Copyright © Gregory Bain, MD)
We hypothesized that the position of the wrist at injury and subsequent position of the carpus initiates the fracture, leading to specific DRF patterns. We retrospectively reviewed a series of CT scans of two-part articular fractures of the distal radius. The DRF and specific points on the scaphoid and lunate were precisely mapped. The images were overlaid, and the proximity of the fracture to these points was measured and statistically analyzed (▶Fig. 1.8).
We used the classification of two-part fractures that Bain et al14 described (▶Fig. 1.9). Each of the defined fractures had an association with various parts of the lunate or scaphoid. For example, radial styloid oblique (RSO) fractures were associated with the volar ulnar aspect of the scaphoid. Intermediate column fractures with the radial border of the lunate. Dorsal ulnar corner (DUC) fractures with the dorsal radial aspect of the lunate. The volar ulnar corner and volar coronal (VC) fractures were associated with the position of the volar lunate and mid-position of the lunate and scaphoid, respectively.
There is an association between the position of the carpal bones and the type of articular fracture that occurs in the distal radius. The vast majority of fractures involve the scapholunate interval. We propose that a compressive load transmitted from the carpus initiates the fracture along the radial aspect of the lunate or the ulnar aspect of the scaphoid. Second, the fracture propagates to the periphery of the articular surface between the sites of ligamentous insertion (▶Fig. 1.10). Wrist position is important; radial deviation causes impaction of the scaphoid, and ulnar deviation—impaction of the lunate. A neutral position will cause corresponding impaction of the scaphoid and lunate.
This theory fits with the work of Pechlaner, who used a cadaveric model to produce a DRF and found concomitant lesions in 63% of cases. The majority of these lesions involved either disruption to the articular disc of the triangular fibrocartilaginous complex or the scapholunate ligament complex.15
There is a high incidence of intercarpal soft tissue injuries (34–54%) in association with DRF although the mechanism and relevance are unclear.16–18 Forward showed that intra-articular fractures were associated with a twofold increase in scapholunate dissociation as seen radiographically at 1 year.16 Mrkonjic reported long-term follow-up of untreated scapholunate injuries in association with DRF. No significant difference in functional scores was found; however, numbers in his series were low, no Geissler grade 4 injuries were included, and most injuries were in the nondominant hand.19
Fig. 1.8(a–e) Representative axial slices of the articular surface and proximal carpal row. Neighboring points on the scaphoid and lunate and a line of best fit are drawn to mark the orientation of the scapholunate joint to the articular surface.(Copyright © Gregory Bain, MD)
We performed a CT study, comparing of two-part DRFs with a control group. The significant increase in the scapholunate distance was noted in fracture subtypes RSO, DUC, sagittal ulnar column, and VC. In particular, both the dorsal and volar aspects of the scapholunate gap were significantly widened in the RSO and DUC groups. This may relate to the level or vector of force at the time of injury (▶Fig. 1.11). The scaphoid, lunate, or both bones as a unit may die-punch the distal radius articular surface, leading to scapholunate rupture and predictable fracture subtypes.
Fig. 1.10 Fracture mechanism of injury. Diagrammatic representation of initiation followed by propagation of articular distal radius fractures. (a) Force is transmitted from the hand—capitate—scaphoid or lunate to the distal radius. (b) Fracture of the articular surface of the radius is initiated at a point along the line of differential load (scapholunate interval). The lunate facet is compressed with ulnar deviation, while the scaphoid is not as loaded. (c) The fracture then propagates to the periphery of the articular surface, preferentially between protected areas of ligamentous insertion.(Copyright © Gregory Bain, MD)
The volar marginal rim of the distal radius contains the lunate fossa and is the “keystone” for loadbearing through the distal radius. The volar rim also contains important volar radiolunate ligament insertions. These act as check reins, preventing volar subluxation and ulnar translocation of the carpus.
As previously described, the fracture can propagate between avulse radiocarpal ligaments. The volar marginal rim fracture represents the failure of the volar radiocarpal ligaments in tension and disruption of these important stabilizers to the rim of the distal radius (▶Fig. 1.12). The radioscaphocapitate (RSC) ligament acts as a secondary stabilizer to the scapholunate joint. Disruption of this ligament may unmask scapholunate injury as diastasis seen on preoperative imaging or under fluoroscopy.
Fig. 1.11 Scapholunate dissociation with different fracture subtypes: sagittal ulnar column (a) and radial styloid oblique (b). Note the position of the wrist (radial or ulnar deviation). The capitate hinges on either the lunate or scaphoid and drives into the scapholunate gap, causing scapholunate ligamentous injury and diastasis. The scaphoid, lunate, or the scapholunate unit impact the distal radius, initiating the fracture.(Copyright © Gregory Bain, MD)
Fig. 1.12(a) The mechanism of injury is often a fall on an outstretched hand, with hyperextension (1), which produces a tension band effect of the volar carpal ligaments (2), producing a localized impaction of the carpus on the radius (3). This causes initiation of a fracture (4) and subsequent fracture propagation (5). The volar plating is designed to provide a buttress of the volar fragments. If these fragments are small or osteoporotic, they can “flow” over the top of the buttress wall (1) and produce a volar collapse of the carpus (2). (b) If the fracture is not reduced and buttressed, the ligaments remain attached to the volar rim fragment, which determines the settling position of the carpus.(Copyright © Gregory Bain, MD)
We performed a retrospective radiological review of 25 volar marginal rim fractures and then compared to a control group of 25 consecutive intra-articular fractures not involving the volar rim.
Volar rim fractures have significantly higher incidence of scapholunate instability (48 vs. 20%), DISI deformity (44 vs. 20%), ulnar styloid fracture (88 vs. 68%), and ulnar translocation of the carpus (20 vs. 8%).
Following fixation, the volar rim fractures had a significantly higher incidence of scapholunate diastasis (48 vs. 20%) and failure of fixation (24 vs. 0%) (▶Fig. 1.13).
Distal radius plate design has evolved to attempt to capture the volar marginal rim fragment. Despite the placement of these plates distal to the watershed line, failure of the carpus can still occur (▶Fig. 1.14). This can be due to very distal comminuted avulsion fragments not being identified at the time of surgery or separation of the ligamentous attachment from the fragment. In these instances, we recommend fragment-specific hook or pin plates (▶Fig. 1.15) and ligament repair or reinforcement through transosseous repair or suture anchor fixation.
Fig. 1.13 Volar rim fractures have a higher incidence of volar instability (white arrow, showing volar subluxation of the carpus), scapholunate instability, and loss of fixation.
Fig. 1.14 Specific volar rim plating. Despite initial capture and support of the lunate fossa fragment, the carpus has subluxated volarly and ulnocarpal translocation has occurred.
Fig. 1.15 Fragment-specific fixation of the volar rim. Note the ligamentous origins on the distal radius in relation to the fracture.(Copyright © Gregory Bain, MD)
We propose a uniting theory incorporating the findings of our studies with the published literature. The fracture event occurs as two distinct stages: fracture initiation followed by propagation.
When the patient falls on the outstretched hand, the wrist is taken past the normal physiological arc of motion. In a dorsally displaced fracture, the volar ligaments and flexor tendons become taut, acting as a tension band, which accentuates the compressive force on the distal radius articular surface.
Force is transmitted from the hand through the midcarpal joint predominantly by the capitate to the proximal carpal row over the scapholunate interval. With the wrist in extension and ulnar deviation, the force is transmitted to the lunate and then the lunate facet. With the wrist in extension and radial deviation, the force is transmitted to the scaphoid and then to the scaphoid facet (▶Fig. 1.10).
When there is disparity between load transmitted across the radiocarpal joint by the scaphoid and the lunate, a line of differential load exists along the scapholunate interval (SLI). Similar to the industrial die-punch analogy where sheet metal fails at the margin of the die, the distal radius subchondral bone plate fails in compression along this line of differential load. In this manner, articular fractures are initiated along the margin of the distal radius corresponding to the scapholunate interval. The scapholunate ligament complex can undergo stretch or complete rupture, depending on the magnitude and direction of force propagation. Intact secondary stabilizers, including the RSC, DRC, DIC, and STT ligaments, will prevent scapholunate diastasis. If there is compromise to these insertion points on the distal radius, such as in the context of a volar marginal fracture, static instability and diastasis will result. The specific point along the SLI ligament depends on a number of variables, including wrist position at the time of injury and both the magnitude and direction of the external force.
The energy of the fracture then dissipates throughout the subchondral bone plate, propagating along the path of least resistance to the periphery of the articular surface, fracturing between protected areas of ligamentous insertion. Predictable articular osteoligamentous fragments are created (▶Fig. 1.8).
In extreme wrist extension, the predominant force on the wrist is tensile over the volar carpal ligament complex. In this case, an avulsion fracture of the volar carpal ligaments will occur, leading to a volar marginal rim fracture. If the wrist is in radial deviation at the time of impact, the ulnar-sided short radiolunate is under most tension and likely to avulse. If the wrist is in ulnar deviation, the long radiolunate or RSC will avulse. After impact, the wrist settles; the lunate flexes and the carpus subluxates volarly (▶Fig. 1.12).
Fracture reduction methods rely on ligamentotaxis, creating a tensile force on the ligaments attached to the fracture fragments. This allows the fragments to realign in their anatomical position. It is imperative when performing surgery on distal radius malunion, therefore to respect this principle and avoid soft tissue stripping of these ligamentous insertions.
DRFs occur in low energy in compromised osteoporotic bone and high energy in younger patients. The distal radius functions as an “arch bridge”; its microarchitecture is a complex evolutionary design and resembles other common engineering structures used to absorb and transmit load safely.
Distal radius follows a predictable sequence of events from initiation through propagation. The bone fails in tension, compression, or both. The fracture pattern is dependent on the vector of force and position of ligaments. Fracture fixation as described in the following chapters should, therefore, respect the pathomechanics of the fracture and the role of the soft tissue stabilizers.
[1] Singh M, Nagrath AR, Maini PS. Changes in trabecular pattern of the upper end of the femur as an index of osteoporosis. J Bone Joint Surg Am 1970;52(3):457–467
[2] Bain GI, MacLean SBM, McNaughton T, Williams R. Microstructure of the Distal Radius and Its Relevance to Distal Radius Fractures. J Wrist Surg 2017;6(4):307–315
[3] Bain GI, MacLean SBM, McNaughton T, Williams R. Erratum: Microstructure of the Distal Radius and Its Relevance to Distal Radius Fractures. J Wrist Surg 2017;6(4):e1–e2
[4] Roth L, Clark A. Understanding Architecture: Its Elements, History, and Meaning. Colorado, USA: Westview Press; 2013
[5] Kuhlmann JN, Luboinski J, Laudet C, et al. Properties of the fibrous structures of the wrist. J Hand Surg [Br] 1990;15(3):335–341
[6] Berger RA. The ligaments of the wrist. A current overview of anatomy with considerations of their potential functions. Hand Clin 1997;13(1):63–82
[7] Berger RA. The anatomy of the ligaments of the wrist and distal radioulnar joints. Clin Orthop Relat Res 2001(383):32–40
[8] Melone CP Jr. Articular fractures of the distal radius. Orthop Clin North Am 1984;15(2):217–236
[9] Melone CP Jr. Open treatment for displaced articular fractures of the distal radius. Clin Orthop Relat Res 1986(202):103–111
[10] Melone CP Jr. Distal radius fractures: patterns of articular fragmentation. Orthop Clin North Am 1993;24(2):239–253
[11] Medoff RJ. Essential radiographic evaluation for distal radius fractures. Hand Clin 2005;21(3):279–288
[12] Mandziak DG, Watts AC, Bain GI. Ligament contribution to patterns of articular fractures of the distal radius. J Hand Surg Am 2011;36(10):1621–1625
[13] Mackenney PJ, McQueen MM, Elton R. Prediction of instability in distal radial fractures. J Bone Joint Surg Am 2006;88(9):1944–1951
[14] Bain GI, Alexander JJ, Eng K, Durrant A, Zumstein MA. Ligament origins are preserved in distal radial intraarticular two-part fractures: a computed tomography-based study. J Wrist Surg 2013;2(3):255–262
[15] Pechlaner S, Kathrein A, Gabl M, et al. [Distal radius fractures and concomitant lesions. Experimental studies concerning the patho-mechanism] Handchir Mikrochir Plast Chir 2002;34(3):150–157
[16] Forward DP, Lindau TR, Melsom DS. Intercarpal ligament injuries associated with fractures of the distal part of the radius. J Bone Joint Surg Am 2007;89(11):2334–2340
[17] Lindau T, Arner M, Hagberg L. Intraarticular lesions in distal fractures of the radius in young adults. A descriptive arthroscopic study in 50 patients. J Hand Surg [Br] 1997;22(5):638–643
[18] Geissler WB, Freeland AE, Savoie FH, McIntyre LW, Whipple TL. Intracarpal soft-tissue lesions associated with an intra-articular fracture of the distal end of the radius. J Bone Joint Surg Am 1996;78(3):357–365
[19] Mrkonjic A, Lindau T, Geijer M, Tägil M. Arthroscopically diagnosed scapholunate ligament injuries associated with distal radial fractures: a 13- to 15-year follow-up. J Hand Surg Am 2015;40(6):1077–1082
Mark Ross, Patrick Groarke
Abstract
This chapter provides guidelines for the radiographic assessment of distal radius fractures. We discuss plain radiographic parameters and their relationship in predicting stability and outcome and determining the adequacy of reduction and fixation strategy where indicated. We will review how to interpret computed tomography in the different planes and the relevance of fragmentation patterns. The value of magnetic resonance imaging and other modalities in assessing the fractured distal radius will be reviewed.
Keywords: radiographs, stability, reduction, parameters, CT, MRI, fragments
Radiographic assessment of the distal radius should be undertaken when the mechanism of injury and presence of deformity or bony tenderness leads to clinical suspicion of a fractured distal radius. Understanding the imaging that needs to be obtained is a key element in the surgeon’s ability to decide on appropriate management. The interpretation of the imaging is an even more important element in planning management, and if fixation is indicated, what fixation method should be employed. It is also critical to understand that each option in the imaging process provides very specific information, which may not be available in other images. In this way, prereduction radiographs (XRs) inform certain aspects, postreduction XRs provide different information, and each plane of computed tomography (CT) imaging is best suited for specific anatomic components of the injury pattern.
The functional demands of each patient differ. As a consequence, the age (physiologic as well as chronologic), employment, and lifestyle of each patient must be used to place the radiographic parameters into context. The goal of treatment is to provide a painless extremity with good function. In surgical decision-making, special attention should be given to the patient’s bone quality. In addition, elderly patients with low demands may tolerate greater variance in many of the radiographic parameters that will be discussed in this chapter. However, with increasing activity level and functional expectations in an aging population, it is increasingly difficult to predict what may be tolerated by elderly patients.1
One of the most challenging considerations is whether the fracture pattern (pre- or postreduction) is stable. That is, is this the position that the fracture will ultimately heal in? The injury (prereduction) XR is very important in determining stability as it provides more information about maximum displacement and the energy of the injury. It is vital that the surgeon considers this series of images when making clinical decisions. Wherever possible, every effort should be made to obtain and assess the injury film. Consideration should also be given to this issue when the fracture has undergone some form of traction or closed reduction prior to initial radiographic assessment as the degree of displacement may be underestimated. A careful history of the interventions following injury is, therefore, vital. Following reduction and institution of closed treatment, it may be possible, with careful attention to cast changes, to control dorsal tilt; however, it is frequent to see the recurrence of radial shortening back to the injury position; thus, this must be considered when deciding on management.
Both increasing age of patients and increasing obesity in younger patients2 have led to an increase in fractures that are comminuted. Comminuted fractures can be difficult to assess on plain XRs, but by applying the same system to evaluate them and with the proper interpretation of the CT, the surgeon can plan the fixation technique where indicated.
Standard XRs are indicated in all suspected distal radius fractures (DRFs). We recommend XR before and after reduction. As noted above, the assessment of stability is best conducted using the injury (prereduction) XRs. Standard views include posteroanterior (PA) and lateral XR. Oblique views can also be helpful in bringing the volar portion of the radial and ulnar aspect of the radius as well as the dorsal ulnar corner (DUC) into view. These provide a two-dimensional image of a three-dimensional (3D) structure but understanding the normal parameters on each view can allow clarification of articular fragments even where CT images are not available. It is important to be able to determine what constitutes a true PA and lateral because many images can be rotated due to patient’s inability to position the arm correctly due to pain, and many parameters are validated in relation to the orthogonal XRs.
The value of these films should not be understated to those carrying out emergent management such as closed manipulation so as to discourage reduction before imaging. This can happen where a grossly deformed wrist presents. Clearly, if the skin is threatened or there are significant neurological symptoms and XRs cannot be obtained emergently, the restoration of gross alignment takes priority; however, careful documentation of the deformity should be made. Injury films can reveal small, nondisplaced, and intra-articular fragments. These fragments might not be visible after anatomical reductions and cast application, which might mask the severity of the fracture. The original displacement and angulation of the fracture can be an indicator of instability. In a study of 406 DRFs, initial displacement was associated with worse QuickDASH score, worse EQ-5D score, reduced grip strength, and reduced range of motion (ROM).3
Although potentially obscured by cast material, these images guide where fragments lie in relation to each other and what type of fixation should be considered. In addition, they guide the adequacy of reduction although should not be used for determining the stability of the fracture and propensity for loss of position (▶Fig. 2.1).
Where nonoperative management is undertaken, follow-up XRs at 1- and 2-week intervals would be considered a minimum and we have observed ongoing loss of position out to 6 weeks and beyond in cast treatment.
CT is more accurate in measuring the change in dorsal angulation over time in DRFs when compared to XRs.4 However, the cost and high dose of radiation are prohibitive in most healthcare systems, and benefits from this increased accuracy have not been clarified.
In severely comminuted fractures, or where the fragments have been inadequately reduced, it might be difficult to establish what the patient’s normal parameters should be. An XR of the uninjured opposite side can be helpful for comparison. Coronal plane translation, as will be discussed later, can be guided by the uninjured side. Contralateral XRs can also give an indication to the energy load to failure of the distal radius by defining normal ulna variance for that patient, given the significant variance in radial length. Increased ulnar variance (loss of radial length) after fracture is also associated with reduced bone mineral density in the distal radius.5
Parameters may vary between populations but the difference is likely to lie within the ranges described below.6
The ulnar border of the ulnar styloid should be continuous with the ulnar border of the shaft. Pronation or supination can result in the ulnar styloid being partially overshadowed by the distal ulna shaft. The radial border of the ulna shaft is concave on a true PA view. Moreover, if the shafts of the radius and ulna are seen to converge, this indicates pronation. A full pronation view has the effect of shortening the radius by at least 0.5 mm.
The PA view presents the carpal facet horizon. This is a radiodense line that represents the volar rim of the lunate facet and medial half of the scaphoid facet, in a radius with normal volar tilt. It extends ulnarward to the sigmoid notch. In a wrist with preserved volar tilt, the XR beam is tangential to the volar half of the articular surface (▶Fig. 2.2). The DUC and dorsal rim are distal to this horizon and less well visualized (▶Fig. 2.3). In a dorsally angulated fracture, the radiodense line will represent the dorsal rim and DUC as the XR beam will be tangential to it (▶Fig. 2.4). The main value of the carpal facet horizon is that a step-off in it will indicate an articular step-off, and understanding whether one is viewing the dorsal or volar half of the joint will help to locate that articular involvement.7
The other implication of the separate representation of the volar and dorsal aspects of the ulnar corner is where to measure ulnar variance and radial inclination from. It has been suggested that true radial length is represented by the average point between the dorsal and volar ulnar corner on the PA view. This may be termed the central reference point (CRP).7 Many studies published up until this point have failed to clarify their method for determining the measuring point for ulnar variance. Perhaps radial length may be a more reproducible measure of true shortening of the radius than ulnar variance because of this factor.
Fig. 2.1(a) Prereduction film demonstrates significant displacement and implies an unstable fracture. (b) Postreduction image demonstrates articular surface involvement more clearly.
Fig. 2.2 The radiograph (XR) beam is tangential to the volar half of the joint in an intact or undisplaced radius.
It should be taken in neutral rotation. On a true lateral XR, the pisiform is located directly over the distal pole of the scaphoid. If the pisiform lies dorsal to the distal pole of the scaphoid, the forearm is rotated into pronation.
Another method of ensuring the wrist is in neutral rotation and is to use the scaphopisocapitate relationship.8 This is described where the rotation of the wrist is set at a position in which the volar margin of the pisiform bone lies at the central third of the interval between the volar cortex of the scaphoid tubercle and volar capitate. Wrist position is also important although may be compromised by pain or deformity in the acute setting. Larsen et al defined the true lateral XR as when the long axis of the radius and third metacarpal bone are collinear in neutral rotation.9
Fig. 2.3 The denser line represents the volar rim when the radius is intact or not dorsally tilted.
Fig. 2.4 In the dorsally tilted radius, the denser line becomes the dorsal rim as it rotates into a tangential relationship to the radiograph (XR) beam.
Fig. 2.5 Carpal alignment with the distal radius is assessed using a line up the volar surface of the radial shaft, which should intersect the center of the head of the capitate.7
Carpal alignment is determined on the lateral. A line extending from the volar surface of the radial shaft (not the metaphysis) should bisect the center of rotation of the proximal pole of the capitate7 (▶Fig. 2.5).
Radiographic parameters are assessed in terms of extra-articular alignment and intra-articular fragmentation/congruence to determine stability and acceptability of reduction.
Fig. 2.6 Significant dorsal comminution implies a higher energy injury that would have had more displacement at the time of injury and may be less stable.
In general terms, comminution is a key extra-articular factor in assessing fracture stability (▶Fig. 2.6
