34,99 €
Ellis and Calne’s Lecture Notes in General Surgery is a comprehensive guide that focuses on the fundamentals of general surgery while systematically covering all the clinical surgical problems that a student or doctor in training may encounter and about which they need to know.
The text includes principles of treatment that have been written at a level to inform the medical student and doctor in training. The text features color illustrations throughout and includes electronic access to a wide range of extra material including case studies, radiological and clinical images, and biographies.
Sample topics discussed in Ellis and Calne’s Lecture Notes in General Surgery include:
Trusted by generations of medical students, including the current authors, the clinical emphasis of Ellis and Calne’s Lecture Notes in General Surgery makes this an essential reference for all those wishing to learn more about general surgery. It is also a perfect revision text for medical students and junior surgeons taking the MRCS examination.
For more information on the complete range of Wiley medical student and junior doctor publishing, please visit: www.wiley.com
To receive automatic updates on Wiley books and journals, join our email list. Sign up today at www.wiley.com/email
This new edition is also available as an e-book. For more details, please see http://www.wiley.com/buy/9781119862482
All content reviewed by students for students
Wiley Medical Education books are designed exactly for their intended audience. All of our books are developed in collaboration with students. This means that our books are always published with you, the student, in mind.
If you would like to be one of our student reviewers, go to www.reviewmedicalbooks.com to find out more.
Sie lesen das E-Book in den Legimi-Apps auf:
Seitenzahl: 1267
Veröffentlichungsjahr: 2022
Cover
Title Page
Copyright Page
Author affiliations
Preface
Acknowledgements
Abbreviations
About the companion website
1 Surgical strategy
History and examination
2 Human factors in surgery
What are human factors?
Error in healthcare
What are the different types of error and failure?
What human factors in surgery should we be thinking about?
3 Fluid and nutrition management
Body fluid compartments
Fluid and electrolyte losses
Normal fluid losses
4 Preoperative assessment
Patient assessment
Management of pre‐existing medical conditions
Operative factors influencing preoperative management
5 Postoperative complications
Classification
Surgical site infection (SSI)
Complications of antibiotic therapy
Venous thromboembolism
Management of the unwell postoperative patient
Postoperative haemorrhage
Postoperative fever
Postoperative respiratory problems
Pulmonary collapse and infection
Postoperative ileus
Abdominal wound dehiscence
Anastomotic leak
Postoperative fistula
Localized intraperitoneal collections
Subphrenic abscess
Pelvic abscess
Delirium
Complications of minimally invasive surgery
Additional resources
6 Acute infections
Cellulitis
Erysipelas
Abscess
Boil
Carbuncle
Specific infections
Necrotizing soft tissue infections
Anthrax
Botulism
Actinomycosis
Additional resources
7 Tumours
Pathology
Clinical features and diagnosis
History
Examination
Special investigations
Tumour markers
Treatment
Anti‐neoplastic agents
Cancer screening
Inherited cancer syndromes
8 Shock
Aetiology
Normal regulation of tissue perfusion
Abnormal regulation of tissue perfusion
Special causes of shock
Sequelae of shock
Principles in the management of patients in shock
Prevention of hypothermia
9 Trauma surgery
Trauma types
The triad of death in trauma
Phase 1: Pre‐hospital trauma care and initial assessment
Phase 2: Initial assessment with ongoing resuscitation
Secondary survey
Damage control ethos
10 Burns
Causes
Severity
The initial management of the severe burn injury patient
Complications
Prognosis
Additional resources
11 The skin and its adnexae
General functions of the skin
Skin anatomy and embryology
Epidermoid cyst
Dermoid cyst
Verruca vulgaris (wart)
Plantar warts
Ganglion
Pilonidal sinus
Hidradenitis suppurativa
Conditions affecting the nails
Tumours of the skin and subcutaneous tissues
Pigmented skin lesions and malignant melanoma
Tumours of sweat glands and sebaceous glands
Adnexal‐derived skin cancers
Vascular anomalies
Lymph vessel tumours
Nerve tumours
Fatty tumours
Additional resources
12 Arterial disease
Arterial trauma
Aneurysm
Abdominal aortic aneurysm
Ruptured abdominal aortic aneurysm
Popliteal aneurysm
Assessing the patient with arterial disease
Lower limb peripheral arterial disease
Acute limb ischaemia (Box 12.3)
Embolism
Atherosclerotic occlusive arterial disease
Coronary occlusive disease
Mesenteric occlusive disease
Carotid artery disease (Figure 12.6)
Raynaud’s disease and Raynaud’s phenomenon
9
Buerger’s disease
Cold injury
Additional resources
13 The heart and thoracic aorta
Introduction
Cardiopulmonary bypass
Cardiac intensive care
Valvular disease
Aortic stenosis
Mitral regurgitation
Mitral stenosis
Ischaemic heart disease
Thoracic aortic disease
14 The chest and lungs
Injury to the chest
Sternal fracture
Fractures of the ribs
Complications
Empyema
Lung tumours
Carcinoid tumours
Lung cancer
Secondary tumours
Additional resources
15 Venous disorders of the lower limb
Normal venous function
Anatomy of the venous drainage of the lower limb
Aetiology of venous disease
Clinical presentation
Assessment of the patient with venous disease
Treatment of venous disease
Deep vein thrombosis
Additional resources
16 The brain and meninges
Space‐occupying intracranial lesions
Intracranial tumours
Intracranial abscess
Intracranial vascular lesions
Hydrocephalus
Additional resources
17 Head injury
Types of injury
Brain injuries
Cerebral perfusion
Management of the patient with a head injury
Indications for surgery in head injuries
Traumatic intracranial bleeding
Other complications
Brain death
Additional resources
18 The spine
Spina bifida (Figure 18.1)
Spinal injuries
Age‐related (degenerative) spinal disorders
Cervical spondylosis
Spinal infection
Spinal tumours
Additional resources
19 Peripheral nerve injuries
Classification
Special investigations
Brachial plexus injuries
Radial nerve injuries (Figure 19.1a)
Median nerve injuries (Figure 19.1b)
Median nerve compression at the wrist (carpal tunnel syndrome)
Ulnar nerve injuries (Figure 19.2)
Ulnar nerve compression at the elbow (cubital tunnel syndrome)
Differential diagnosis of flexion deformities of the fingers
Sciatic nerve injuries
Common peroneal nerve injuries
Lateral cutaneous nerve of the thigh compression: meralgia paraesthetica
Cervical sympathetic nerve injuries: Horner’s syndrome
8
Additional resources
20 The oral cavity
The oral cavity
Embryology
Congenital disease of the oral cavity and lips
Acquired disease of the oral cavity and lips
Non‐malignant disease of oral mucosa and oral cavity
Lesions of the oral mucosa including lip mucosa
Potential malignant disorders of the mouth
Intraoral pigmented lesions
Intraoral lumps
Malignant disease of the cutaneous lip
Malignant disease of oral mucosa and oral cavity
Reconstructive surgery in head and neck cancer
Additional resources
21 The salivary glands
Inflammation
Viral infections
Acute bacterial parotitis
Chronic recurrent parotid sialadenitis
Sjögren's syndrome
Calculi
Salivary tumours
Adenolymphoma
Carcinoma
Additional resources
22 The oesophagus
Dysphagia
Perforations of the oesophagus
Caustic stricture of the oesophagus
Achalasia of the cardia
Plummer–Vinson syndrome
6
Oesophageal diverticula
Pharyngeal pouch
Reflux oesophagitis
Tumours of the oesophagus
Carcinoma
Barrett’s oesophagus
10
and adenocarcinoma
Additional resources
23 The stomach and duodenum
Congenital hypertrophic pyloric stenosis
Duodenal atresia
Peptic ulcer
The acute peptic ulcer
The chronic peptic ulcer
Perforated peptic ulcer
Pyloric stenosis
Gastrointestinal haemorrhage
Bleeding peptic ulcer
Gastroparesis
Gastric volvulus
Gastric tumours
Gastric polyps
Gastrointestinal stromal tumours
Gastric lymphoma
Gastroenteropancreatic neuroendocrine tumours (GEP‐NETs)
Gastric carcinoma
Additional resources
24 The surgery of obesity
Obesity
25 The small intestine
Meckel’s diverticulum
Crohn’s disease
Tumours of the small intestine
Neuroendocrine tumours
Additional resources
26 The appendix
Embryology and anatomy
Acute appendicitis
Clinical features
Appendiceal abscess
Appendix mass (Box 26.2)
Appendiceal neoplasms
Additional resources
27 The colon
Constipation and diarrhoea
Diverticular disease
Angiodysplasia
Colitis
Tumours
Colostomy
Additional resources
28 The rectum and anal canal
Bright red rectal bleeding ( Table 28.1)
Haemorrhoids
Anal fissure
Anorectal abscesses
Anal fistula
Stricture of the anal canal
Prolapse of the rectum
Pruritus ani
Faecal incontinence
Tumours
Rectal polyps
Carcinoma of the rectum
Anal cancer
Additional resources
29 The acute abdomen
Aetiology
Clinical assessment
Special investigations
Principles of treatment
Non‐surgical treatment
Surgery
Peritonitis
Special causes of peritonitis
Mesenteric ischaemia
30 Intestinal obstruction and paralytic ileus
Mechanical obstruction
Closed loop obstruction
Adhesive obstruction
Volvulus
Neonatal intestinal obstruction
Distal intestinal obstruction syndrome
Intussusception
Paralytic ileus
Pseudo‐obstruction
Additional resources
31 Hernia
Definition
Abdominal wall hernias
Inguinal hernia
Anatomy (Figure 31.2)
Femoral hernia
Richter’s hernia
Umbilical hernia
Congenital umbilical hernia
Paraumbilical hernia
Divarication of the recti
Epigastric hernia
Incisional hernia
Differential diagnosis of midline hernia
Unusual hernias
Diaphragmatic hernias
Traumatic diaphragmatic hernias
Acquired hiatal hernias
Reflux oesophagitis
Additional resources
32 The liver
Liver enlargement
Jaundice
Congenital abnormalities
Liver trauma
Acute infections of the liver
Hydatid disease of the liver
Cirrhosis
The effects of liver failure
Hepatorenal syndrome
Liver neoplasms
Cholangiocarcinoma
Liver surgery
Additional resources
33 The gallbladder and bile ducts
Congenital anomalies
Cholelithiasis (gallstones)
Bile composition and function
Gallbladder physiology
Gallstone types
Clinical manifestations of gallstones
Gallbladder polyps
Carcinoma of the gallbladder
Cholangiocarcinoma
Additional resources
34 The pancreas
Congenital anomalies
Annular pancreas
Heterotopic pancreas
Acute pancreatitis
Severe acute pancreatitis
Chronic pancreatitis
Pancreatic cysts
Pancreatic tumours
Pancreatic neuroendocrine tumours
Insulinoma ( β‐cell tumour)
Gastrinoma (Zollinger–Ellison syndrome,
11
non‐β‐cell islet tumour)
Pancreatic carcinoma
Additional resources
35 The spleen
Splenomegaly
Splenectomy
Ruptured spleen
Additional resources
36 The lymph nodes and lymphatics
The lymphadenopathies
Lymphoedema
Additional resources
37 The breast
Developmental anomalies
Symptoms of breast disease
Traumatic fat necrosis
Acute inflammation of the breast (mastitis)
Chronic inflammatory conditions of the breast
Benign breast disease
Gynaecomastia
Phyllodes tumour
Carcinoma of the breast
Paget’s disease of the nipple
Inflammatory breast cancer
Patients unfit for surgery
Metastatic disease
Carcinoma of the male breast
Breast screening
Additional resources
38 The neck
Branchial cyst and sinus
Tuberculous cervical adenitis
Carotid body tumour (chemodectoma)
Additional resources
39 The thyroid
Congenital anomalies
Lingual thyroid
Thyroglossal cyst
Thyroid physiology
Pathology of goitre
Clinical features in thyroid disease
Hyperthyroidism
Hypothyroidism
Hashimoto’s disease
Riedel’s thyroiditis
De Quervain’s thyroiditis
Investigations in thyroid disease
Clinical classification of thyroid swellings
Outline of treatment of goitre
Hyperthyroidism
Complications of thyroidectomy
Thyroid tumours
Additional resources
40 The parathyroids
Anatomy and development
Physiology
Effects of increased PTH production
Hypoparathyroidism
Hyperparathyroidism
Additional resources
41 The thymus
Tumours
Myasthenia gravis
42 The adrenal glands
Physiology
Enzyme disorders
Cushing’s syndrome
Primary hyperaldosteronism (Conn’s syndrome)
The adrenogenital syndromes (Figure 42.1)
Non‐functioning tumours of the adrenal cortex
Adrenocortical carcinoma
Adrenomedullary tumours
Ganglioneuroma
Adrenal ‘incidentaloma’
Hypertension
Renovascular hypertension
Additional resources
43 The kidney and ureter
Congenital anomalies
Common renal anomalies (Figure 43.2)
Horseshoe kidney
Duplex system
Polycystic disease
Renal cysts
Haematuria
Injury to the kidney
Hydronephrosis
Urinary tract calculi
Urinary tract infections
Reflux nephropathy
Pyonephrosis
Renal abscess
Renal tuberculosis
Renal failure
Management
Acute tubular necrosis
Chronic renal failure
Renal tumours
Nephroblastoma (Wilms’ tumour
5
)
Renal cell carcinoma
Tumours of the renal pelvis and ureter
Additional resources
44 The bladder
Congenital anomalies
Urachal anomalies
Bladder exstrophy (ectopia vesicae)
Rupture of the bladder
Diverticulum of the bladder
Bladder stone
Bladder tumours
Urothelial carcinoma
Additional resources
45 The prostate
Benign prostatic enlargement
Urinary retention
Bladder neck obstruction
Prostatitis
Prostate cancer
Additional resources
46 The male urethra
Anatomy
Congenital anomalies
Urethral injury and trauma in the male
Urethral stricture
47 The penis
Phimosis
Paraphimosis
Balanitis
Penile cancer
Erectile dysfunction
Additional resources
48 The testis and scrotum
Abnormalities of testicular descent
Scrotal swelling
Epididymal cysts
Hydrocoele
Acute infections of the testis and epididymis
Chronic infections of the testis
Torsion of the testis
Varicocoele
Disorders of the scrotal skin
Tumours of the testis
Male infertility
Additional resources
49 Transplantation surgery
Historical background
Classification of grafts
Organ donors
Exclusions to organ donation
Organ preservation
Organ recipients
The immunology of organ transplantation
Organ matching
Rejection
Principles of immunosuppressive therapy
Complications of transplantation
Results of clinical organ transplantation
Additional resources
Index
End User License Agreement
Chapter 1
Table 1.1 Examples of important facts to determine in patients with pain an...
Table 1.2 Common prefixes and suffixes used in surgery
Chapter 2
Table 2.1 Simplified Human Factors Analysis and Classification System (HFAC...
Table 2.2 Situations that might increase the chance of an error in the oper...
Table 2.3 Items to consider during a team briefing
Chapter 3
Table 3.1 Normal daily fluid losses
Table 3.2 Electrolyte content of intravenous fluids
Chapter 4
Table 4.1 Factors involved in the estimation of risk using P‐POSSUM
Table 4.2 The ASA grading system
Chapter 5
Table 5.1 Postoperative complications following abdominal surgery
Table 5.2 The Two‐Level Wells score for estimating the clinical probability...
Table 5.3 Causes of postoperative fever according to postoperative day
Chapter 7
Table 7.1 The 10 most common cancer killers in the UK in 2018
Table 7.2 Tumour markers
Chapter 9
Table 9.1 ATMIST Communication acronym
Table 9.2 The primary survey
Table 9.3 Blood tests for trauma patients
Chapter 10
Table 10.1 Burn comparison
Chapter 11
Table 11.1 Fitzpatrick skin types
Table 11.2 Ten‐year survival according to Breslow depth
Chapter 23
Table 23.1 Summary of Recommendations for gastroduodenal ulcer disease
Chapter 24
Table 24.1 Estimated increased risk for the obese of developing associated ...
Chapter 27
Table 27.1 Diverticulum terminology
Table 27.2 Crohn’s colitis and ulcerative colitis*
Chapter 28
Table 28.1 Rectal bleeding
Chapter 31
Table 31.1 Characteristic differences that help differentiate indirect and ...
Chapter 32
Table 32.1 Diagnosis of jaundice
Chapter 37
Table 37.1 Pathologic TNM staging for breast cancer, AJCC UICC 2010
11
Chapter 45
Table 45.1 Risk stratification for localized or locally advanced prostate c...
Chapter 1
Figure 1.1 Location of referred pain for the abdominal organs.
Figure 1.2 Example of how to record abdominal examination findings.
Chapter 2
Figure 2.1 The Swiss cheese model of human error. Each slice can act as a ba...
Figure 2.2 The WHO surgical safety checklist.
Chapter 3
Figure 3.1 Estimated electrolyte concentration in body fluids encountered po...
Chapter 5
Figure 5.1 (a–d) Progression of deep vein thrombosis. PE, pulmonary embolus....
Figure 5.2 The anatomy of the subphrenic spaces (sagittal views): (a) right ...
Chapter 8
Figure 8.1 Starling law.
Figure 8.2 Anatomical & physiological assessment and treatment of shock.
Chapter 9
Figure 9.1 The triad of death in trauma care.
Figure 9.2 Phases of damage control. DCS: damage control surgery; ICU: inten...
Chapter 10
Figure 10.1 A partial‐thickness burn (a) leaves part or the whole of the ger...
Figure 10.2 The Lund and Browder chart allows more accurate estimation of bu...
Figure 10.3 The ‘rule of nines’ – a useful guide to the estimation of the ar...
Chapter 11
Figure 11.1 Layers of the skin with adnexal structures.
Figure 11.2 (a) The normal skin contains melanocytes (shown as cells) and me...
Chapter 12
Figure 12.1 (a–c) Types of aneurysms.
Figure 12.2 Aortic repairs.
Figure 12.3 Classification of endoleaks.
Figure 12.4 Tracings of arteriograms. (a) An example of a good ‘run‐off’ fro...
Figure 12.5 Source of peripheral emboli. DVT, deep vein thrombosis.
Figure 12.6 Consequences and treatment of carotid artery stenosis.
Figure 12.7 Different measurement techniques for determining the degree of c...
Chapter 13
Figure 13.1 Cardiopulmonary bypass.
Figure 13.2 A saphenous vein graft from the aorta to the right coronary arte...
Figure 13.3 (a) A persistent ductus arteriosus – note its close relationship...
Figure 13.4 Stanford classification of aortic dissection.
Chapter 14
Figure 14.1 Flail chest. On inspiration, the detached segment of the chest w...
Figure 14.2 Tension pneumothorax produced by a valvular tear in the lung. Ai...
Figure 14.3 Underwater seal chest drain in the treatment of a pneumothorax. ...
Chapter 15
Figure 15.1 The venous system of the leg. Note that there are two main super...
Figure 15.2 Normal veins and incompetent varicose veins. Note that the vein ...
Chapter 16
Figure 16.1 The common sites of intracranial aneurysms.
Figure 16.2 The ventricular system.
Chapter 17
Figure 17.1 Coup and contre‐coup injuries – mechanism.
Figure 17.2 Decerebrate and decorticate postures.
Figure 17.3 (a) Extradural haematoma and (b) acute subdural haematoma. The l...
Chapter 18
Figure 18.1 Spina bifida.
Figure 18.2 The three‐column spine concept.
Figure 18.3 The relationship of the spinal cord and nerve roots to the verte...
Chapter 19
Figure 19.1 (a) Radial nerve injury: wrist drop, together with anaesthesia o...
Figure 19.2 Ulnar nerve injury:
main en griffe
with anaesthesia of the ulnar...
Figure 19.3 Horner’s syndrome.
Chapter 20
Figure 20.1 The oral cavity and oropharynx viewed from the front.
Figure 20.2 The ventral aspect of a foetal head showing the three prominence...
Chapter 22
Figure 22.1 (a–e) Barium swallow appearances of common causes of dysphagia....
Figure 22.2 A pharyngeal pouch emerging between the two components of the in...
Chapter 23
Figure 23.1 Ramstedt’s pyloromyotomy. The thickened muscle at the pylorus is...
Figure 23.2 The principal operations once commonly performed for peptic ulce...
Figure 23.3 Techniques to close a perforated ulcer.
Figure 23.4 The upper panel (a) illustrates a total gastrectomy with a Roux‐...
Chapter 24
Figure 24.1 Relative risk of death from all causes according to body mass in...
Figure 24.2 The three common bariatric surgical procedures.
Chapter 26
Figure 26.1 The positions in which the appendix may lie, together with their...
Figure 26.2 The blood supply of the appendix.
Chapter 27
Figure 27.1 The relationship of diverticula of the colon to the taenia coli ...
Figure 27.2 Typical colonic operations. For a lesion in the right colon, a r...
Chapter 28
Figure 28.1 Distribution of different conditions around the anal canal.
Figure 28.2 The anatomy of anorectal abscesses.
Figure 28.3 The anatomy of anal fistulas.
Figure 28.4 Staging of rectal cancer by modified Dukes’ and TNM classificati...
Figure 28.5 Surgical procedures for carcinoma of rectum.
Chapter 31
Figure 31.1 The differences between (a) a reducible, (b) an irreducible and ...
Figure 31.2 The anatomy of the inguinal canal: (a) with the external oblique...
Figure 31.3 The anatomy of the femoral canal and its surrounds to show the r...
Figure 31.4 The relationships of an indirect inguinal and a femoral hernia c...
Figure 31.5 The development of the diaphragm. The drawing shows the four con...
Figure 31.6 (a) Sliding hiatus hernia: the stomach and lower oesophagus slid...
Chapter 32
Figure 32.1 The metabolism of bilirubin.
Figure 32.2 The sites of occurrence of portal–systemic communications in pat...
Figure 32.3 (a–c) Segmental anatomy of the liver showing inflow vessels (hep...
Chapter 33
Figure 33.1 Developmental anomalies of the gallbladder. (a) A long cystic du...
Figure 33.2 Biliary anatomy and anomalies. In the majority of individuals, t...
Figure 33.3 The varieties of gallstones.
Figure 33.4 Obstructive jaundice due to stone is usually associated with a s...
Chapter 34
Figure 34.1 (a–d) The development of the pancreas and biliary tree.
Figure 34.2 Whipple’s pancreaticoduodenectomy. (a) The initial appearance ch...
Chapter 37
Figure 37.1 Anatomical features of the breast.
Figure 37.2 The different origins of
in situ
and invasive ductal and lobular...
Figure 37.3 The lymphatic drainage of the breast.
Chapter 38
Figure 38.1 The derivatives of the branchial pouches and clefts.
Chapter 39
Figure 39.1 The descent of the thyroid, showing possible sites of ectopic th...
Figure 39.2 The relationship of the recurrent laryngeal nerve to the thyroid...
Chapter 42
Figure 42.1 Adrenocortical steroid synthesis
Figure 42.2 Scheme for investigation of incidental adrenal lesion on imaging...
Figure 42.3 Mechanism of renal hypertension.
Chapter 43
Figure 43.1 Development of the pro‐, meso‐ and metanephric systems (after La...
Figure 43.2 Renal abnormalities: (a) polycystic kidney; (b) Horseshoe kidney...
Figure 43.3 Some important causes of bleeding in the urinary tract.
Figure 43.4 Diagram of the effects of urinary calculi.
Chapter 44
Figure 44.1 Embryological development of the bladder and lower urinary tract...
Chapter 45
Figure 45.1 The clinical staging of prostatic carcinoma.
Chapter 48
Figure 48.1 Questions to resolve the differential diagnosis of a scrotal swe...
Figure 48.2 (a–d) The anatomical classification of hydrocoeles (the ring at ...
Cover Page
Title Page
Copyright Page
Author affiliations
Preface
Acknowledgements
Abbreviations
About the companion website
Table of Contents
Begin Reading
Index
WILEY END USER LICENSE AGREEMENT
iii
iv
vi
vii
viii
ix
x
xi
xii
xiii
xiv
1
2
3
4
5
6
7
8
9
10
11
12
13
14
15
16
17
18
19
20
21
22
23
24
25
26
27
28
29
30
31
32
33
34
35
36
37
38
39
40
41
42
43
44
45
46
47
48
49
50
51
52
53
54
55
56
57
58
59
60
61
62
63
64
65
66
67
68
69
70
71
72
73
74
75
76
77
78
79
80
81
82
83
84
85
86
87
88
89
90
91
92
93
94
95
96
97
98
99
100
101
102
103
104
105
106
107
108
109
110
111
112
113
114
115
116
117
118
119
120
121
122
123
124
125
126
127
128
129
130
131
132
133
134
135
136
137
138
139
140
141
142
143
144
145
146
147
148
149
150
151
152
153
154
155
156
157
158
159
160
161
162
163
164
165
166
167
168
169
170
171
172
173
174
175
176
177
178
179
180
181
182
183
184
185
186
187
188
189
190
191
192
193
194
195
196
197
198
199
200
201
202
203
204
205
206
207
208
209
210
211
212
213
214
215
216
217
218
219
220
221
222
223
224
225
226
227
228
229
230
231
232
233
234
235
236
237
238
239
240
241
242
243
244
245
246
247
248
249
250
251
252
253
254
255
256
257
258
259
260
261
262
263
264
265
266
267
268
269
270
271
272
273
274
275
276
277
278
279
280
281
282
283
284
285
286
287
288
289
290
291
292
293
294
295
296
297
298
299
300
301
302
303
304
305
306
307
308
309
310
311
312
313
314
315
316
317
318
319
320
321
322
323
324
325
326
327
328
329
330
331
332
333
334
335
336
337
338
339
340
341
342
343
344
345
346
347
348
349
350
351
352
353
354
355
356
357
358
359
360
361
362
363
364
365
366
367
368
369
370
371
372
373
374
375
376
377
378
379
380
381
382
383
384
385
386
387
388
389
390
391
392
393
394
395
396
397
398
399
400
401
402
403
404
405
406
407
408
409
410
411
412
413
414
415
416
417
418
419
420
421
422
423
424
425
426
427
428
429
430
431
432
433
434
435
436
437
438
439
440
441
442
443
444
445
446
447
448
449
450
451
452
453
454
455
456
457
458
459
460
461
462
463
464
465
466
467
468
469
470
471
472
473
474
475
476
477
478
479
480
481
482
483
484
485
486
487
488
489
490
491
492
493
494
495
496
497
498
499
500
501
502
503
504
505
506
507
508
509
510
511
512
513
Edited by
Christopher Watson, MD BChir FRCS
Professor of Transplantation and Honorary Consultant Surgeon
Cambridge University Hospitals NHS Foundation Trust
Cambridge, UK
Justin Davies, MA MChir FRCS (Gen Surg)
FEBS (Coloproctology)
Consultant Colorectal Surgeon
Cambridge University Hospitals NHS Foundation Trust
Affiliated Assistant Professor
University of Cambridge
Fellow in Clinical Medicine
Downing College, Cambridge, UK
Fourteenth Edition
This fourteenth edition first published 2023© 2023 John Wiley & Sons Ltd
Edition History1965, 1968, 1970, 1972, 1977, 1983, 1987, 1993, 1998, 2002, 2006, 2011, 2016 by Harold Ellis, Sir Roy Y. Calne and Christopher J. E. Watson
All rights reserved. No part of this publication may be reproduced, stored in a retrieval system, or transmitted, in any form or by any means, electronic, mechanical, photocopying, recording or otherwise, except as permitted by law. Advice on how to obtain permission to reuse material from this title is available at http://www.wiley.com/go/permissions.
The right of Christopher Watson, and Justin Davies to be identified as the authors of the editorial material in this work has been asserted in accordance with law.
Registered OfficeJohn Wiley & Sons, Inc., 111 River Street, Hoboken, NJ 07030, USAJohn Wiley & Sons Ltd, The Atrium, Southern Gate, Chichester, West Sussex, PO19 8SQ, UK
For details of our global editorial offices, customer services, and more information about Wiley products visit us at www.wiley.com.
Wiley also publishes its books in a variety of electronic formats and by print‐on‐demand. Some content that appears in standard print versions of this book may not be available in other formats.
Trademarks: Wiley and the Wiley logo are trademarks or registered trademarks of John Wiley & Sons, Inc. and/or its affiliates in the United States and other countries and may not be used without written permission. All other trademarks are the property of their respective owners. John Wiley & Sons, Inc. is not associated with any product or vendor mentioned in this book.
Limit of Liability/Disclaimer of WarrantyThe contents of this work are intended to further general scientific research, understanding, and discussion only and are not intended and should not be relied upon as recommending or promoting scientific method, diagnosis, or treatment by physicians for any particular patient. In view of ongoing research, equipment modifications, changes in governmental regulations, and the constant flow of information relating to the use of medicines, equipment, and devices, the reader is urged to review and evaluate the information provided in the package insert or instructions for each medicine, equipment, or device for, among other things, any changes in the instructions or indication of usage and for added warnings and precautions. While the publisher and authors have used their best efforts in preparing this work, they make no representations or warranties with respect to the accuracy or completeness of the contents of this work and specifically disclaim all warranties, including without limitation any implied warranties of merchantability or fitness for a particular purpose. No warranty may be created or extended by sales representatives, written sales materials or promotional statements for this work. This work is sold with the understanding that the publisher is not engaged in rendering professional services. The advice and strategies contained herein may not be suitable for your situation. You should consult with a specialist where appropriate. The fact that an organization, website, or product is referred to in this work as a citation and/or potential source of further information does not mean that the publisher and authors endorse the information or services the organization, website, or product may provide or recommendations it may make.Further, readers should be aware that websites listed in this work may have changed or disappeared between when this work was written and when it is read. Neither the publisher nor authors shall be liable for any loss of profit or any other commercial damages, including but not limited to special, incidental, consequential, or other damages.
Library of Congress Cataloging‐in‐Publication Applied forPaperback ISBN: 9781119862482
Cover Design: WileyCover Image: © Morsa Images/Getty Images
Hemantha AlawattegamaConsultant AnaesthetistCambridge University Hospitals NHS Foundation Trust
Anita BalakrishnanConsultant HPB SurgeonCambridge University Hospitals NHS Foundation Trust
Peter A. BrennanConsultant Maxillofacial Surgeon and Honorary Professor of SurgeryPortsmouth Hospitals University TrustPortsmouth
Alexandra J. ColquhounConsultant UrologistCambridge University Hospitals NHS Foundation Trust
Aman Singh CoonarConsultant Thoracic SurgeonRoyal Papworth HospitalCambridge
Patrick CoughlinLeeds Vascular InstituteLeeds
Justin DaviesConsultant Colorectal SurgeonCambridge University Hospitals NHS Foundation TrustAffiliated Assistant ProfessorUniversity of CambridgeFellow in Clinical MedicineDowning College, Cambridge
Amer J. DurraniConsultant Plastic & Reconstructive SurgeonCambridge University Hospitals NHS Foundation Trust
Brian FishConsultant ENT/Head and Neck SurgeonCambridge University Hospitals NHS Foundation Trust
Manj GohelConsultant Vascular & Endovascular SurgeonCambridge University Hospitals NHS Foundation TrustHonorary Senior LecturerImperial CollegeLondon
Stavros GourgiotisConsultant SurgeonCambridge Oesophago‐Gastric CentreCambridge University Hospitals NHS Foundation Trust
Ian GrantConsultant Plastic SurgeonCambridge University Hospitals NHS Foundation Trust
Ekpemi IruneConsultant Laryngology, Head & Neck Surgeon Cambridge University Hospitals NHS Foundation Trust
David P. JenkinsConsultant Cardiothoracic SurgeonRoyal Papworth HospitalCambridge
Eleftheria KleidiConsultant Oncoplastic Breast SurgeonCambridge University Hospitals NHS Foundation Trust
Vasilis KosmoliaptsisHonorary Consultant Abdominal Transplant, HPB and Endocrine SurgeonAssociate Professor, Department of SurgeryUniversity of Cambridge
Rodney J. C. LaingConsultant NeurosurgeonCambridge University Hospitals NHS Foundation Trust
Arthur McPheeConsultant UrologistCambridge University Hospitals NHS Foundation Trust
Kanwalraj MoarConsultant Cleft and Maxillofacial SurgeonCambridge University Hospitals NHS Foundation Trust
Jonathan MortonConsultant Colorectal SurgeonCambridge University Hospitals NHS Foundation Trust
Ioanna G. PanagiotopoulouConsultant Colorectal SurgeonCambridge University Hospitals NHS Foundation Trust
Raaj Kumar PraseedomConsultant HPB & Transplant SurgeonCambridge University Hospitals NHS Foundation Trust
Stephen PriceHonorary Consultant NeurosurgeonCambridge University Hospitals NHS Foundation Trust
Christopher PringConsultant Bariatric SurgeonSt Richard’s HospitalChichesterVisiting ProfessorUniversity of Surrey
Peter SafranekConsultant SurgeonCambridge Oesophago‐Gastric CentreCambridge University Hospitals NHS Foundation Trust
Constantinos SimillisConsultant Colorectal SurgeonCambridge University Hospitals NHS Foundation Trust
Lynsey SpillmanHepatology and Liver Transplant DietitianCambridge University Hospitals NHS Foundation Trust
Vijay SujendranConsultant SurgeonCambridge Oesophago‐Gastric CentreCambridge University Hospitals NHS Foundation Trust
Elizabeth TweedleConsultant Colorectal SurgeonCambridge University Hospitals NHS Foundation Trust
Christopher WatsonProfessor of Transplantation and Honorary Consultant SurgeonCambridge University Hospitals NHS Foundation Trust
Many medical students will have effectively written their own textbook at the end of their clinical course – a digest of the lectures and tutorials assiduously attended and of the textbooks meticulously read. Unfortunately, students may approach the qualifying examinations burdened by the thought of many pages of excellent and exhaustive textbooks wherein lies the wisdom required of them by the examiners. Although the Internet is increasingly used as a source of information, we believe that there is still a serious need for a book that briefly sets out the important facts in General Surgery that are classified, analysed and, as far as possible, rationalized for the revision student. These lecture notes represent such a text; they are in no way a substitute for the standard textbooks, but they do draw together, in a logical way, the fundamentals of General Surgery and its subspecialties. As such we hope it will also be of use to the junior surgeon.
We wish to point the reader to the electronic resource accompanying the text, including case studies, radiographs and histology slides illustrating common conditions mentioned in the text, as well as a quiz to test the reader’s knowledge.
The first edition of Lecture Notes in General Surgery was written by Harold Ellis and Sir Roy Calne and published in 1965. This is the first edition without their involvement, and we acknowledge their huge contribution to surgery and its teaching. Surgery has changed enormously since the days of that first edition, and each subsequent edition has tried to keep pace with surgical practice. Never has the pace of change been so great as it is today. The biggest changes have been seen in the development of specialist services, at the expense of the generalist, and the development of multidisciplinary teams to optimize management. Recognizing this, previous editions have relied heavily on colleagues from other specialties to keep chapters updated and relevant. For this edition we have taken this further, and each chapter now has a nominated expert author – who, for some chapters, has updated previous content; and, for others, has written completely new content – to ensure relevance for today’s student.
Christopher WatsonJustin Davies
We are grateful to our colleagues – senior and junior doctors, and students – who have read and critiqued this text during its production, and to many readers and reviewers for their constructive criticisms. We are indebted to those colleagues in years gone by, too numerous to mention here, who have developed the text upon which the new chapter experts have built. We would also like to acknowledge the continued help give by the staff at Wiley for seeing this project through to publication, in particular James Watson, Catriona King, Mandy Collison, Ella Elliott, and Indirakumari Siva.
ABPI
ankle brachial pressure index
ABG
arterial blood gas
ABLS
Advanced Burns Life Support
ACE
angiotensin‐converting enzyme
ACTH
adrenocorticotrophic hormone
ADH
antidiuretic hormone
ADT
androgen deprivation therapy
AFP
α‐fetoprotein
AIs
aromatase inhibitors
AIDS
acquired immune deficiency syndrome
AIN
anal intraepithelial neoplasia
AJCC
American Joint Committee on Cancer
ALK
anaplastic lymphoma kinase
ALP
alkaline phosphatase
ALT
alanine transaminase
ANC
axillary node clearance
ANS
axillary node sampling
ANUG
acute necrotizing ulcerative gingivitis
APFC
acute peripancreatic fluid collection
APACHE
Acute Physiology and Chronic Health Evaluation
APTT
activated partial thromboplastin time
APUD
amine precursor uptake and decarboxylation
ASA
American Society of Anesthesiologists
ASD
atrial septal defect
ASIA
American Spinal Injury Association
AST
aspartate transaminase
ATN
acute tubular necrosis
ATLS
Advanced Trauma Life Support
AXR
abdominal X‐ray
β‐HCG
β‐human chorionic gonadotrophin
BCG
bacille Calmette–Guérin
BCS
breast conserving surgery
BMI
body mass index
BPH
benign prostatic hyperplasia
CABG
coronary artery bypass graft
CAPOX
capecitabine and oxaliplatin
CAR
chimeric antigen receptor
CaSR
calcium sensing receptor
CEA
carcinoembryonic antigen
CEAP
Clinical, Etiological, Anatomical and Pathophysiological
CHRPE
congenital hypertrophy of the retinal pigment epithelium
CMV
cytomegalovirus
CNS
central nervous system
COPD
chronic obstructive pulmonary disease
CPAP
continuous positive airways pressure
CPE
carbapenemase‐producing enterobacteriaceae
CPPS
chronic pelvic pain syndrome
CRE
carbopenem‐resistant enterobacteriaceae
CRP
C‐reactive protein
CSF
cerebrospinal fluid
CT
computed tomography
CTLA4
cytotoxic lymphocyte–associated antigen 4
CTPA
computed tomographic pulmonary angiography
CVP
central venous pressure
CXR
chest X‐ray
DBD
donation after brain‐stem death
DCD
donation after circulatory death
DCIS
ductal carcinoma
in situ
DCS
damage control surgery
DDAVP
deamino‐
D
‐arginine vasopressin
DHCA
deep hypothermic circulatory arrest
DIC
disseminated intravascular coagultion
DIOS
distal intestinal obstruction syndrome
DMSA
dimercaptosuccinic acid
DOPA
dihydroxyphenyl alanine
DST
dexamethasone suppression test
DTC
differentiated thyroid cancer
DTPA
diethylene triamine penta‐acetic acid
DVT
deep venous thrombosis
EBV
Epstein‐Barr virus
ECG
electrocardiogram
ECST
European Carotid Surgery Trial
EGFR
epidermal growth factor receptor
EGC
early gastric cancer
EMG
electromyography
EMR
endoscopic mucosal resection
EMSB
Emergency Management of Severe Burns
ER
oestrogen receptor
ERAS
Enhanced Recovery After Surgery
ERCP
endoscopic retrograde cholangiopancreatography
ESBL
extended spectrum β‐lactamase
ESD
endoscopic submucosal dissection
ESR
erythrocyte sedimentation rate
ESWL
extracorporeal shock‐wave lithotripsy
EUS
endoscopic or endoluminal ultrasound
EVAR
Endovascular Aneurysm Repair
5‐FU
5‐fluorouracil
FAP
familial adenomatous polyposis
FAST
focused abdominal sonography for trauma
FBC
full blood count
FDG
fluorodeoxyglucose
FEV
1
forced expiratory volume in 1 second
FFP
fresh frozen plasma
FIT
faecal immunochemical test
FNAC
fine‐needle aspiration cytology
FOLFOX
folinic acid and oxaliplatin
FSH
follicle‐stimulating hormone
GABA
γ‐aminobutyric acid
GANT
gastrointestinal autonomic nervous tumour
GCS
Glasgow Coma Score
GEP‐NETs
gastroenteropancreatic neuroendocrine tumours
GFR
glomerular filtration rate
GGT
γ‐glutamyl transferase
GI
gastrointestinal
GIM
gastrointestinal metaplasia
GIST
gastrointestinal stromal tumour
GLA
γ‐linolenic acid
GOJ
gastro‐oesophageal junction
GnRH
gonadotrophin‐releasing hormone
GORD
gastroesophageal reflux disease
GPA
granulomatosis with polyangiitis
GTN
glyceryl trinitrate
HAART
highly active antiretroviral treatment
HALO
haemorrhoidal artery ligation
HALT
hungry, anxious/angry, late, tired
HAMN
high‐grade appendiceal mucinous neoplasms
HbA1c
glycosylated haemoglobin
HCl
hydrochloric acid
HCC
hepatocellular carcinoma
HER2
human epidermal growth factor receptor 2
HGD
high‐grade dysplasia
HHT
hereditary haemorrhagic telangiectasia
HHV
human herpes virus
HIPEC
heated intraperitoneal chemotherapy
HIV
human immunodeficiency virus
HLA
human leucocyte antigen
HNPCC
hereditary non‐polyposis colon cancer
HoLEP
holmium laser prostatectomy
HPOA
hypertrophic pulmonary osteoarthropathy
HPV
human papilloma virus
HQIP
Healthcare Quality Improvement Partnership
HRT
hormone replacement therapy
HSV
herpes simplex virus
HTIG
human tetanus immunoglobulin
IBD‐U
inflammatory bowel disease unclassified
ICC
interstitial cell of Cajal
ICP
intracranial pressure
ICSI
intracytoplasmic sperm injection
ICU
intensive care unit
IFN
interferon
IMA
inferior mesenteric artery
IMV
inferior mesenteric vein
INR
International normalized ratio
IPMN
intraductal papillary mucinous neoplasm
IPSS
International prostate symptom score
ITU
intensive therapy unit
IVC
inferior vena cava
IVF
in vitro
fertilization
IVU
intravenous urogram
JVP
jugular venous pressure
KSHV
Kaposi sarcoma herpes virus
KUB
kidneys, ureters and bladder
LAD
left anterior descending artery
LAMN
low‐grade appendiceal mucinous neoplasms
LCIS
lobular carcinoma
in situ
LDH
lactate dehydrogenase
LGD
low‐grade dysplasia
LHRH
luteinizing hormone‐releasing hormone
LIF
left iliac fossa
LiDCO
transpulmonary lithium dilution cardiac output
LMWH
low‐molecular‐weight heparin
LUTS
lower urinary tract symptoms
MAG3
m
ercapto‐
a
cetyl tri
g
lycine
MAMC
midarm muscle circumference
MCN
mucinous cystic neoplasm
MDT
multidisciplinary team
MELD
model for end‐stage liver disease
MEN
multiple endocrine neoplasia
MHC
major histocompatibility complex
MIBG
meta‐iodobenzylguanidine
MIBI
methoxyisobutylisonitrile
MOI
mechanism of injury
mpMRI
multiparametric MRI
MR
magnetic resonance
MRC
Medical Research Council
MRCP
magnetic resonance cholangiopancreatography
MRI
magnetic resonance imaging
MRSA
methicillin‐resistant
Staphylococcus aureus
mTOR
mechanistic target of rapamycin
MUST
Malnutrition Universal Screening Tool (ch 3)
NAFLD
non‐alcoholic fatty liver disease
NASCET
North American Symptomatic Carotid Endarterectomy Trial
NCEPOD
National Confidential Enquiry into Perioperative Death
NELA
National Emergency Laparotomy Audit
NEWS
National Early Warning Score
NEN
neuroendocrine neoplasms
NHS
National Health Service
NICE
National Institute of Health and Care Excellence
NG
nasogastric
NOACs
novel oral anticoagulants
NPI
Nottingham Prognostic Index
NSAIDs
non‐steroidal anti‐inflammatory drugs
NSCLC
non‐small cell lung cancer
NSGCT
non‐seminomatous germ cell tumour
NST
no special type
OCP
oral contraceptive pill
OPG
orthopantomogram
OPSI
overwhelming post‐splenectomy infection
PAC
plasma aldosterone concentration
PBC
primary biliary cholangitis
PCI
percutaneous coronary intervention
pcr
pathologic complete response
PDE5
phosphodiesterase type 5
PDGFA
platelet‐derived growth factor receptor α
PDL
programmed death ligand
PE
pulmonary embolism
PEC
percutaneous endoscopic colostomy
PEG
polyethylene glycol
PEG
percutaneous endoscopic gastrostomy
PET
positron emission tomography
PI_RADS
prostate imaging reporting and data system
PICC
peripherally inserted central catheter
PN
parenteral nutrition
PNET
primitive neuroectodermal tumour
POEM
per oral endoscopic myotomy
POSSUM
Physiological and Operative Severity Score for the enUmeration of Mortality and Morbidity
PPE
personal protective equipment (ch 2)
PPGL
Phaeochromocytoma and paraganglioma
PPP
patient, procedure and people
PR
progesterone
PRA
plasma renin activity
PRC
plasma renin concentration
PSA
prostate‐specific antigen
PSC
primary sclerosing cholangitis
PT
prothrombin time
PTA
percutaneous transluminal angioplasty
PTC
percutaneous transhepatic cholangiography
PTCA
percutaneous transluminal coronary angioplasty
PTFE
polytetrafluoroethylene
PTH
parathormone
PUJ
pelviureteric junction
PV
portal vein
RIF
right iliac fossa
SAH
subarachnoid haemorrhage
SBP
spontaneous bacterial peritonitis
SCLC
small‐cell lung cancer
SDHX
succinate dehydrogenase subunit genes
SDM
shared decision‐making
SGA
Subjective Global Assessment
SGOT
serum glutamic oxaloacetic transaminase (synonymous with AST)
SGPT
serum glutamic pyruvic transaminase (synonymous with ALT)
SIADH
syndrome of inappropriate antidiuretic hormone
SLE
systemic lupus erythematosus
SLN
sentinel lymph node
SMA
superior mesenteric artery
SMV
superior mesenteric vein
SORT
Surgical Outcome Risk Tool
SSI
surgical site infection
SV
splenic vein
SVT
superficial vein thrombosis
TAD
targeted axillary dissection
TB
tuberculosis
T3
tri‐iodothyronine
T4
tetra‐iodothyronine, thyroxine
TACE
transarterial chemoembolization
TAE
tumour embolisation
TAP
transversus abdominus plane
TAVI
transaortic valve implantation
TCC
transitional cell carcinoma
TED
thromboembolism deterrent
TEVAR
thoracic endovascular aortic repair
TIA
transient ischaemic attack
TIPS
transjugular intrahepatic portosystemic shunt
TNF
tumour necrosis factor
TNM
tumour node metastasis
TOE
transoesophageal echocardiography
TPA
tissue plasminogen activator
TPN
total parenteral nutrition
TRAM
transverse rectus abdominis myocutaneous
TRH
thyrotrophin‐releasing hormone
TRUS
transrectal ultrasound
TSH
thyroid‐stimulating hormone
TUR
transurethral resection
UC
urothelial carcinoma
UC
ulcerative colitis
UFH
unfractionated heparin
UKELD
United Kingdom Model for end‐stage liver disease
UW
University of Wisconsin
VAB
vacuum‐assisted biopsy
VAC
vacuum‐assisted closure
VATS
video‐assisted thoracoscopic surgery
VAWCM
vacuum‐assisted wound closure device with mesh‐mediated fascial traction
VEGF
vascular endothelial growth factor
VEGFR‐3
vascular endothelial growth factor receptor 3
VET
venous thromboembolism
VIP
vasoactive intestinal polypeptide
VISA
vancomycin‐intermediate
Staphylococcus aureus
VRE
vancomycin‐resistant
Enterococcus
VRSA
Vancomycin‐resistant
Staphylococcus aureus
WHO
World Health Organisation
This book is accompanied by a companion website.
www.wiley.com/go/Watson/GeneralSurgery14
The website features:
Interactive multiple choice and short‐answer questions
Case Studies
Extra images and photographs
Biographies
Justin Davies
To understand the principles of taking a clear history, performing an appropriate examination, presenting the findings and formulating a management plan for diagnosis and subsequent investigations and treatment.
To understand the common nomenclature used in surgery.
The principles of assessing patients referred to a surgical team has changed little in recent times. These include:
Taking an accurate history
.
Examination of the patient
.
Accurate and contemporaneous documentation
(
written and/or electronic)
.
Constructing a differential diagnosis
. Ask the question ‘What diagnoses would best explain this clinical picture?’
Special investigations
. Which laboratory and imaging tests are required to confirm or refute the clinical diagnosis?
Management
. Decide on the management of the patient, including provision of adequate analgesia. Remember that this will include reassurance, explanation, and good communication skills.
Development of clinical skills is of paramount importance in all aspects of medicine and surgery. In some circumstances, excessive reliance on special investigations and extensive imaging may be unnecessary. It is important to remember that the patient may be apprehensive and will often be in pain, especially when presenting as an emergency. Attending to these issues is an especially important aspect of good clinical care.
The history should be an accurate reflection of what the patient has said. It is important to ask open questions such as ‘When were you last well?’ and ‘What happened next?’ rather than closed questions such as ‘Do you have chest pain?’ If you have a positive finding, it is important to explore this further with more directed questioning, for example, ‘When did it start?’ ‘What makes it better, and what makes it worse?’ ‘Where did it start and where did it go?’ ‘Did it come and go, or was it constant?’ If the symptom is characterized by bleeding, ask about what sort of blood (e.g. fresh, bright red, dark red), when it started, how much, whether there were clots, whether it was mixed in with food/faeces and whether it was associated with pain. Remember that most patients come to see a general surgeon, particularly in the emergency setting, because of abdominal pain or bleeding (Table 1.1). You will need to find out as much as you can about the presenting symptoms.
Keep in mind that the patient may have little accurate anatomical knowledge. They might say ‘my stomach hurts’, but this may be due to lower chest or periumbilical pain – it is important to ask them to point to the site of the pain. Bear in mind that they may be pointing to a site of referred pain, and a vague description such as ‘back pain’ will need further exploration and clarification as to where it is in the back – the sacrum or lumbar, thoracic or cervical spine, or possibly the loin or subscapular regions. Exploring pain outside of the abdomen is important, particularly shoulder pain. This may, for example, suggest referred pain from the diaphragm or gallbladder.
Table 1.1 Examples of important facts to determine in patients with pain and rectal bleeding
Pain
Rectal bleeding
Exact site
Estimation of amount (often inaccurate)
Radiation
Timing of bleeding
Length of history
Colour – bright red, dark red, black
Periodicity
Accompanying symptoms – pain, vomiting (haematemesis)
Nature – constant/colicky
Associated features – fainting, shock, etc.
Severity
Blood mixed in stool, lying on surface, on toilet paper, in toilet bowl
Relieving and aggravating factors
Accompanying features (e.g. jaundice, vomiting, haematuria)
It is often useful to consider the viscera in terms of their embryology. Thus, epigastric pain is generally from foregut structures such as the stomach, duodenum, liver, gallbladder, spleen and pancreas; periumbilical pain is midgut pain from the small bowel and ascending colon, including the appendix; suprapubic pain is hindgut pain, originating in the colon, rectum and other structures of the cloaca such as the bladder, uterus and Fallopian tubes (Figure 1.1). Testicular pain may also be periumbilical, reflecting the intra‐abdominal origin of these organs before their descent into the scrotum – this is exemplified by the child with testicular torsion who initially complains of pain in the centre of their abdomen.
Figure 1.1 Location of referred pain for the abdominal organs.
Remember the classic quartet in this order:
Inspection.
Palpation.
Percussion.
Auscultation.
Careful inspection is always time well spent. Inspect the patient generally, as to how they lie and breathe. Are they tachypnoeic because of a chest infection or in response to a metabolic acidosis? Look at the patient’s hands and feel the pulse. Asking the patient to walk may be revealing in someone with claudication or in assessment of general fitness.
Only after careful inspection should palpation start. If you are examining the abdomen in the emergency setting, it is important to ask the patient to cough. This is a surrogate test of rebound tenderness and indicates where the site of inflammation is within the peritoneal cavity. It is often helpful to examine the ‘normal’ or non‐symptomatic side first, be it the abdomen, hand, leg or breast. Look carefully at the patient’s face while you palpate, as this may provide subtle clues regarding discomfort or tenderness. If there is a lump, decide which anatomical plane it lies within. Is it in the skin, in the subcutaneous tissue, in the muscle layer or, in the case of the abdomen, in the underlying cavity? Is the lump pulsatile, expansile or mobile?
We practice in an era where electronic patient records are becoming more commonplace, although currently the UK still has the majority of hospitals with paper‐based medical records. The number with electronic records will continue to increase over time.
Always write or type your findings completely and accurately in a contemporaneous fashion. Start by recording the date and time of the assessment and check that you have the correct patient’s notes open. Record all the negative as well as positive findings. Avoid abbreviations where possible since they may mean different things to different people; for example, PID – you may mean pelvic inflammatory disease, but the next person might interpret it as a prolapsed intervertebral disc. Use the appropriate surgical terminology (Table 1.2).
Table 1.2 Common prefixes and suffixes used in surgery
Prefix
Related organ/structure
angio‐
blood vessels
arthro‐
a joint
cardio‐
heart
cholecysto‐
gallbladder
coelio‐
peritoneal cavity
colo‐ and colon‐
colon
colpo‐
vagina
cysto‐
urinary bladder
gastro‐
stomach
hepato‐
liver
hystero‐
uterus
laparo‐
peritoneal cavity
mammo‐ and masto‐
breast
nephro‐
kidney
oophoro‐
ovary
orchid‐
testicle
rhino‐
nose
thoraco‐
chest
Suffix
Procedure
‐centesis
surgical puncture, often accompanied by drainage, e.g. thoracocentesis
‐desis
fusion, e.g. arthrodesis
‐ectomy
surgical removal, e.g. colectomy
‐oscopy
visual examination, usually through an endoscope, e.g. laparoscopy
‐ostomy
creating a new opening (mouth) on the surface, e.g. colostomy
‐otomy
surgical incision, e.g. laparotomy
‐pexy
surgical fixation, e.g. orchidopexy
‐plasty
to mould or reshape, e.g. angioplasty; also to replace with prosthesis, e.g. arthroplasty
‐rrhaphy
surgically repair or reinforce, e.g. herniorrhaphy
Figure 1.2 Example of how to record abdominal examination findings.
Illustrate your examination unambiguously with simple drawings when possible – use anatomical reference points and measure the diameter of any lumps accurately. When drawing abdominal findings, use a hexagonal representation (Figure 1.2). A continuous line implies an edge; shading can represent an area of tenderness or the site where pain is experienced. If you can feel all around a lump, draw a line to indicate this; if you can feel only the upper margin, show only this. Annotate the drawings with your findings (Figure 1.2). At the end of your notes, write a single paragraph summary and make a diagnosis or record a differential diagnosis. Outline a management plan and state what investigations should be done, indicating those which you have already arranged. Sign your notes and print your name, position and contact details, with the time and date recorded.
The purpose of presenting a case is to convey to your colleagues the salient clinical features, diagnosis or differential diagnosis, management, and investigations of the patient. The presentation should ideally be succinct and to the point, containing important positive and negative findings. At the end of a case presentation, the listening team should have an excellent word picture of the patient and their problems, what needs to be monitored and what plans you have for management.
Peter A. Brennan
To understand the factors that can affect safe surgical practice.
To know what measures to take to mitigate risks in surgery.
There are many definitions of this term, but a simple one to remember is how we interact with each other (in teams), the systems in which we work, our variability and the factors that affect our performance and those of team members. In healthcare, human factors application can lead to improved patient safety, better team working and staff morale. Important elements of human factors also include situational awareness, effective team working, safe and effective communication, and good leadership. Furthermore, by recognizing how both physical and mental performance deteriorate over time helps to consolidate their importance.
As humans, we regularly make mistakes, with an average of five to seven simple errors affecting each of us every day. These might be something simple such as forgetting a wallet or a mobile phone when leaving for work because of a distraction. While these errors or omissions might be annoying, error in healthcare is a cause of significant patient harm and mortality. We can never completely eliminate error, and as the above‐mentioned examples demonstrate, it is a familiar part of everyday life. The term ‘never event’ has been coined to describe occurrences that should not occur in a healthcare setting and includes wrong site surgery, retained instruments and swabs, and incorrect naso‐gastric tube placement. A ‘never event’ is somewhat of a misnomer as error can never be completely eliminated, but the chances of error occurring can be minimized.
The Roman Philosopher Cicero (106–43 BC) wrote ‘anyone is liable to err (make a mistake), but only a fool persists in error’. Learning from mistakes and sharing lessons widely with others is one of the most important elements to improving patient safety across healthcare.
The interplay of human error and human factors in clinical incidents (including factors that have their origins in hospitals where we work) is becoming more widely understood. It is often more than one issue (or layer) that leads to error, and this is readily demonstrated by the well‐known Swiss cheese model (Figure 2.1).
Often, factors are multifactorial and take place simultaneously – recognizing this fact is the first step to understanding human factors in surgery. These multifactorial issues include ones that affect us as individuals, such as tiredness, repetition, stress, the effects of distraction and multi‐tasking. Other factors can occur as part of team working, and these include poor communication or leadership, loss of situational awareness, and steep (or flat) authority gradients. The introduction of the World Health Organisation (WHO) surgical checklist (Figure 2.2) has improved attitudes towards pre‐surgery briefing and patient safety, and the benefits of recognizing and applying these human factors principles in surgery are well known.
Figure 2.1 The Swiss cheese model of human error. Each slice can act as a barrier.
One in 20 hospital admissions has some form of error, and of these one in 20 are serious (i.e. one in 4,000 admissions). The operating theatre is known to be one of the most potentially error‐prone places in the hospital as a result of high patient turnover, site‐specific treatments, many heterogeneous surgical procedures, staff limitations and unfamiliar teams.
The Human Factors Analysis and Classification System (HFACS) categorizes failure across four broad domains:
Organizational influences.
Unsafe supervision.
Preconditions to unsafe acts.
Unsafe acts.
All four domains can be applied to the Swiss cheese model (Figure 2.1). Failures at each level may be active (decisions, actions or attitudes by individuals or surgical teams) or latent (results of deficiencies with the hospital or management team). Examples of failures in the clinical setting that can have their origin in the employing organization include pressures of overbooked clinics or operating theatre sessions, meeting clinical and hospital targets and prolonged working hours without breaks. Medical error may, therefore, begin to develop well before the actual event itself (such as wrong site surgery) as a result of institutional failure.
Table 2.1 summarizes some important human factors that can contribute to and ultimately lead to surgical error, as categorized by the HFACS. These include factors that affect us as individuals, such as tiredness and fatigue; nutritional status; emotional states, including anger and stress; multi‐tasking; and loss of situational awareness. These will now be considered further:
Commercial aviation recognizes how tiredness and fatigue can influence personal performance and increase the likelihood of accidents, and as a result, rules are in place for maximum work hours. Tiredness (a state that can only be reversed by sleep) and fatigue (more complex in its aetiology and can be the result of chronic tiredness and/or physical or mental exhaustion) are both found in surgical team members. Both
