43,99 €
The word ‘addiction’ these days is used to refer to a chronic condition where there is an
unhealthily powerful motivation to engage in a particular behaviour. This can be driven by
many different factors – physiological, psychological, environmental and social. If we say that it
is all about X, we miss V, W, Y and Z. So, some people think addicts are using drugs to escape
from unhappy lives, feelings of anxiety and so on; many are. Some people think drugs become
addictive because they alter the brain chemistry to create powerful urges; that is often true.
Others think that drug taking is about seeking after pleasure; often it is. Some take the view that
addiction is a choice – addicts weigh up the pros and cons of doing what they do and decide
the former outweigh the latter. Yet others believe that addicts suffer from poor impulse control;
that is often true… And so it goes on.
When you look at the evidence, you see that all these positions capture important aspects of
the problem – but they are not complete explanations. Neuroscience can help us delve more
deeply into some of these explanations, while the behavioural and social sciences are better at
exploring others. We need a model that puts all this together in a way that can help us decide
what to do in different cases. Should we prescribe a drug, give the person some ‘tender loving
care’, put them in prison or what? Theory of Addiction provides this synthesis.
The first edition was well received:
‘Throughout the book the reader is exposed to a vast number of useful observations...The
theoretical aims are timely, refreshing, ambitious and above all challenging. It opens up a new
way of looking at addiction and has the potential to move the field of addiction a considerable
leap forward. Thus we wholeheartedly would like to recommend the book for students as well
as scholars. Read and learn!’ Nordic Studies on Alcohol and Drugs
‘The book provides a comprehensive review of existing theories - over 30 in all - and this
synthesis of theories constitutes an important contribution in and of itself... West is to be
commended for his synthesis of addiction theories that span neurobiology, psychology and
social science and for his insights into what remains unexplained.’ Addiction
Sie lesen das E-Book in den Legimi-Apps auf:
Seitenzahl: 563
Veröffentlichungsjahr: 2013
Cover
Series
Title
Copyright
Preface
Chapter 1: Introduction: journey to the centre of addiction
Preparing for the journey
In the end
What this book does
The synthetic theory of addiction in brief
References
Chapter 2: Definition, theory and observation
Defining addiction (addiction is not an elephant)
Diagnosing and measuring addiction
Theory and supposition
‘Big observations’ in the field of addiction
Recapitulation
References
Chapter 3: Beginning the journey: addiction as choice
Addiction as a reflective choice
Irrational, ill-informed choice and unstable preferences
Addiction as the exercise of choice based on desires
References
Chapter 4: Choice is not enough: the concepts of impulse and self-control
Reports of feelings of compulsion
Powerful motives versus impaired control
Personality and addiction typologies
Self-efficacy
The transition from lapse to relapse
Impulse control
Self-regulation as a broadly based concept
Urges and craving
Addiction as a failure of self-control over desires and urges
References
Chapter 5: Addiction, habit and instrumental learning
Instrumental learning
Mechanisms underpinning instrumental learning
Classical conditioning
More complex learning models
Social learning
Associative learning
References
Chapter 6: Addiction in populations, and comprehensive theories
Addiction in populations
Comprehensive theories of addiction
What is addiction and how can we explain it?
References
Chapter 7: Development of a comprehensive theory
A functional classification of theories of addiction
Addiction as reflective choice
Addiction as irrational choice
Addiction, compulsion and self-control
Addiction, instrumental learning and habit
Addiction, choice, compulsion and habit
References
Chapter 8: A synthetic theory of motivation
Understanding behaviour in context: the COM-B model
Focus on motivational theory
The human motivational system
Structure and function of the human motivational system
The ‘head model’
Momentum and inertia
Adaptation: ways in which experience affects motivational disposition
The ‘representational system’, consciousness and dual process models
Self and self-control
Mental effort and motivational resources
What motivates us
The unstable mind
A summary: key propositions from PRIME theory
References
Chapter 9: A theory of addiction
Addiction is
The pathologies underlying addiction
A return to some ‘big observations’ about addiction
The abnormalities underlying addiction
Effects of interventions
Recommendations and predictions regarding addiction interventions
Testing the theory
First results
Conclusions
References
Index
End User License Agreement
Chapter 2: Definition, theory and observation
Table 2.1 Commonly used definitions of addiction/dependence
Table 2.2 Examples to test a definition of addiction
Table 2.3 Diagnostic criteria for addiction
Table 2.4 Drugs and activities that at least some people have seriously claimed to have addictive potential
Chapter 7: Development of a comprehensive theory
Table 7.1 Categories of theories of addiction together with examples
Table 7.2 Key concepts used in theories of addiction (adapted from West 2013)
Chapter 3: Beginning the journey: addiction as choice
Figure 3.1 A simplified view of the Opponent Process Model of addiction. The affective (hedonic or emotional) response to a stimulus (such as a drug) is the underlying a-process, which elicits the opponent b-process. These processes add together to create the pleasant A-state followed by an unpleasant B-state. On first use the pleasant A-state is large, followed by a small B-state (see i). With repeated drug use, the b-process increases in magnitude and duration, leading to an experience dominated by the unpleasant symptoms associated with withdrawal (see ii).
Figure 3.2 Identity Shift Theory (adapted from Kearney and O'Sullivan 2003).
Chapter 5: Addiction, habit and instrumental learning
Figure 5.1 Location of the mesolimbic dopamine pathway (adapted from Tomkins and Sellers 2001).
Figure 5.2 Drug actions of the mesolimbic dopamine pathway (adapted from Tomkins and Sellers 2001).
Figure 5.3 White's Multiple Memory System Theory of addiction (reproduced from White 1996).
Figure 5.4 Changes occurring in development of addiction according the stimulus–response (S–R) Habit Learning Model (top) and the Incentive Sensitisation Theory (bottom). According to the Habit Learning Model, addiction is primarily due to the development of strong S–R habits (indicated by the thick arrow from a drug cue, the conditioned stimulus (CS), to a response). According to the incentive sensitisation view, the primary change is in the ability of drug cues to create a sensitised motivational response of incentive salience which then leads to compulsive drug pursuit (thick arrow) via an unconditioned stimulus (UCS) (Robinson and Berridge [2003]. Modified with permission from Annual Review of Psychology).
Chapter 6: Addiction in populations, and comprehensive theories
Figure 6.1 Consequences of conflict from the Excessive Appetites Model (adapted from Orford 2001).
Figure 6.2 The three pathways to pathological gambling (adapted from Blaszczynski and Nower 2002).
Chapter 7: Development of a comprehensive theory
Figure 7.1 Classification of theories of addiction (West 2013).
Figure 7.2 Addiction as rational choice. Given the opportunity, the individual makes decisions to seek out or enact the behaviour depending on analysis of the costs and benefits. A high frequency or intensity of activity simply reflects the preferences of the individual given current circumstances. Increases in frequency or intensity derive from feedback from the behaviour to the costs and benefits of engaging in it.
Figure 7.3 A Choice Theory of addiction with self-control added.
Figure 7.4 A simplified diagram of a Choice Theory of addiction with self-control, and instrumental learning added.
Chapter 8: A synthetic theory of motivation
Figure 8.1 The COM-B system for analysing behaviour in context.
Figure 8.2 A schematic of the five subsystems that make up the human motivational system.
Figure 8.3 Subsystems and their main paths of influence in the PRIME Theory of motivation.
Figure 8.4 The ‘head’ model of the human motivational system
Figure 8.5 A representation of PRIME theory with the different constructs labelled as either reflective or automatic.
Figure 8.6 Examples of different trajectories in the unstable mind.
Figure 8.7 Example of the epigenetic landscape proposed by Waddington (1977) to represent how genes and environment interact in ontogenesis.
Chapter 9: A theory of addiction
Figure 9.1 The SNAP model of smoking cessation. Transitions occur as result of a combination of tension and triggers. Tension is a feeling of a desire to change (want or need) arising from a combination of dissatisfaction with current situation or hope that change can be achieved. Triggers are events that momentarily either raise the tension (including increasing urgency), reduce barriers, simply put the idea of change in the person's mind or show a pathway by which the change can occur.
Cover
Table of Contents
Begin Reading
Cover
Contents
iii
ii
iv
ix
x
1
2
3
4
5
6
7
8
9
10
11
12
13
14
15
16
17
18
19
20
21
22
23
24
25
26
27
28
29
30
31
32
33
34
35
36
37
38
39
40
41
42
43
44
45
46
47
48
49
50
51
52
53
54
55
56
57
58
59
60
61
62
63
64
65
66
67
68
69
70
71
72
73
74
75
76
77
78
79
80
81
82
83
84
85
86
87
88
89
90
91
92
93
94
95
96
97
98
99
100
101
102
103
104
105
106
107
108
109
110
111
112
113
114
115
116
117
118
119
120
121
122
123
124
125
126
127
128
129
130
131
132
133
134
135
136
137
138
139
140
141
142
143
144
145
146
147
148
149
150
151
152
153
154
155
156
157
158
159
160
161
162
163
164
165
166
167
168
169
170
171
172
173
174
175
176
177
178
179
180
181
182
183
184
185
186
187
188
189
190
191
192
193
194
195
196
197
198
199
200
201
202
203
204
205
206
207
208
209
210
211
212
213
214
215
216
217
218
219
220
221
222
223
224
225
226
227
228
229
230
231
232
233
234
235
236
237
238
239
240
241
242
243
244
245
246
247
248
249
250
251
252
253
254
255
256
257
258
259
260
261
262
263
Addiction Press aims to communicate current ideas and evidence in this expanding field, not only to researchers and practising health professionals, but also to policy makers, students and interested non-specialists. These publications are designed to address the significant challenges that addiction presents to modern society.
Other books in the Addiction Press series
Clinical Handbook of Adolescent Addiction
Edited by R. Rosner
978 0 4709 7234 2
Harm Reduction in Substance Use and High-Risk Behaviour
Edited by R. Pates & D. Riley
978 1 4051 8297 3
Injecting Illicit Drugs
Edited by R. Pates, A. McBride & K. Arnold
978 1 4051 1360 1
Neuroimaging in Addiction
Edited by B. Adinoff & E. Stein
978 0 4706 6014 0
Treating Drinkers and Drug Users in the Community
T. Waller & D. Rumball
978 0 6320 3575 5
Understanding and Treating Addictions: Psychological Assessment and Intervention
Edited by A. Copello
978 1 4051 2417 1
Addiction: Evolution of a Specialist Field
Edited by G. Edwards
978 0 6320 5976 8
Second Edition
Robert West
Jamie Brown
University College London
This edition first published 2013, © 2013 by John Wiley & Sons, Ltd. First edition published 2009 © 2009 Robert West
Registered office: John Wiley & Sons, Ltd, The Atrium, Southern Gate, Chichester, West Sussex, PO19 8SQ, UK
Editorial offices: 9600 Garsington Road, Oxford, OX4 2DQ, UK The Atrium, Southern Gate, Chichester, West Sussex, PO19 8SQ, UK 111 River Street, Hoboken, NJ 07030-5774, USA
For details of our global editorial offices, for customer services and for information about how to apply for permission to reuse the copyright material in this book please see our website at www.wiley.com/wiley-blackwell
The right of the author to be identified as the author of this work has been asserted in accordance with the UK Copyright, Designs and Patents Act 1988.
All rights reserved. No part of this publication may be reproduced, stored in a retrieval system, or transmitted, in any form or by any means, electronic, mechanical, photocopying, recording or otherwise, except as permitted by the UK Copyright, Designs and Patents Act 1988, without the prior permission of the publisher.
Designations used by companies to distinguish their products are often claimed as trademarks. All brand names and product names used in this book are trade names, service marks, trademarks or registered trademarks of their respective owners. The publisher is not associated with any product or vendor mentioned in this book. It is sold on the understanding that the publisher is not engaged in rendering professional services. If professional advice or other expert assistance is required, the services of a competent professional should be sought.
The contents of this work are intended to further general scientific research, understanding, and discussion only and are not intended and should not be relied upon as recommending or promoting a specific method, diagnosis, or treatment by health science practitioners for any particular patient. The publisher and the author make no representations or warranties with respect to the accuracy or completeness of the contents of this work and specifically disclaim all warranties, including without limitation any implied warranties of fitness for a particular purpose. In view of ongoing research, equipment modifications, changes in governmental regulations, and the constant flow of information relating to the use of medicines, equipment, and devices, the reader is urged to review and evaluate the information provided in the package insert or instructions for each medicine, equipment, or device for, among other things, any changes in the instructions or indication of usage and for added warnings and precautions. Readers should consult with a specialist where appropriate. The fact that an organization or Website is referred to in this work as a citation and/or a potential source of further information does not mean that the author or the publisher endorses the information the organization or Website may provide or recommendations it may make. Further, readers should be aware that Internet Websites listed in this work may have changed or disappeared between when this work was written and when it is read. No warranty may be created or extended by any promotional statements for this work. Neither the publisher nor the author shall be liable for any damages arising herefrom.
Library of Congress Cataloging-in-Publication Data
West, Robert, 1955– Theory of addiction / Robert West and Jamie Brown. – Second edition. pages cm Includes bibliographical references and index. ISBN 978-0-470-67421-5 (pbk. : alk. paper) 1. Substance abuse. 2. Compulsive behavior. I. Brown, Jamie, 1984– II. Title. RC564.W47 2014 616.86–dc23 2013013746
A catalogue record for this book is available from the British Library.
Wiley also publishes its books in a variety of electronic formats. Some content that appears in print may not be available in electronic books.
Cover image: www.istockphoto.com/stock_shoppeCover design by www.hisandhersdesign.co.uk
The first edition of Theory of Addiction summarised the major theoretical approaches that attempt to explain the range of phenomena that we call ‘addiction’ and synthesise these into an overarching theory that captured their main core features. It was borne out of a need for a single model that could account for features of addiction that involve conscious choice, powerful desires and impulses, and attempts at, and failure of, self-control. It needed to bring in ideas of pleasure-seeking as well as relief from mental and physical discomfort. It needed to account for individual differences in susceptibility to addiction and explain when and why many addicts ‘recover’ without help but also why some interventions help some addicts. It needed to explain why population-level interventions such as price increases and reductions in availability can influence the prevalence of addiction and why some medicines can help addicts recover without appearing to provide any of the functions provided by the addictive behaviour.
Given that motivation lay at the heart of addiction it became apparent that a theory of addiction needed to be built on a theory of motivation, and that involved creating a synthetic model of motivation. A first draft of a theory was constructed, aiming to be as parsimonious as possible, while still accounting for the full range of ‘big observations’. The goal was not only to explain, but also to generate hypotheses about how one might intervene more effectively to combat addiction. It was always intended that the theory would continue to develop, both in order to account for more observations and to make it more useful when it comes to designing interventions.
Since the first edition was published, new findings have accumulated and experience has been gathered in communicating the theory. After 6 years, it seemed appropriate to update the book to take account of this. Important revisions include the following:
1.
Updating the definition and conceptualisation of addiction.
2.
Updating descriptions of theories in the literature.
3.
Providing a functional classification of theories and models of addiction.
4.
Providing a description of core concepts involved in these theories.
5.
Updating the description of the synthetic theory of addiction (PRIME Theory) to improve clarity and take account of new observations.
6.
Assessing PRIME Theory in the light of new evidence.
7.
Setting the PRIME Theory motivation in the context of a more general theory of behaviour that also incorporates capability and opportunity.
The theory is still very much ‘work in progress’. There is much to be done to fill in gaps, test assumptions and examine how far the theory adds value when designing interventions.
This book aims to take you on a journey. The starting point is the simplest possible common-sense theory of addiction and the destination will be a theory that accounts for the available evidence on how addiction develops, who becomes addicted, what they become addicted to and how some of them recover. The path is traced by exploring with successive versions of the theory how it needs to be extended or changed to take account of the available evidence. The goal is to arrive at a theory that is comprehensive yet parsimonious, coherent, and above all useful. It aims to stimulate research and to guide clinicians and policy makers in coming up with better ways of tackling this global scourge. This is a continuing journey. As new evidence emerges and better ways of explaining the evidence are brought to light, the theory will need updating.
The field of addiction is not short on theories. There are psychological theories, biological theories, sociological theories, economic theories, biopsychosocial theories and more. Almost all of the theories in the field of addiction capture important elements of the phenomenon. The problem is that each theory seems to stem from an idea or set of ideas that accounts for a part of the problem but does not account for other features that were previously addressed by other theories. They view addiction from a particular perspective and focus on what is immediately visible from that point of view. They neglect key features that are visible from other points of view. This militates against developing an ‘incremental science’ of addiction in which new theoretical principles build on what has gone before, correcting areas where they fail to account for data and creating new avenues for exploration.
Lesen Sie weiter in der vollständigen Ausgabe!
Lesen Sie weiter in der vollständigen Ausgabe!
Lesen Sie weiter in der vollständigen Ausgabe!
Lesen Sie weiter in der vollständigen Ausgabe!
Lesen Sie weiter in der vollständigen Ausgabe!
Lesen Sie weiter in der vollständigen Ausgabe!
Lesen Sie weiter in der vollständigen Ausgabe!
Lesen Sie weiter in der vollständigen Ausgabe!
Lesen Sie weiter in der vollständigen Ausgabe!
Lesen Sie weiter in der vollständigen Ausgabe!
